M. Nita
Molecular Biology · 2016
A researcher in the Science corpus: 1 paper; trusts 4 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 21 citations into it
- Physiology — 8 citations into it
- Neurology — 6 citations into it
- Clinical Biochemistry — 5 citations into it
Whom M. Nita trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- M. Brownlee
- E. Stadtman
- W. Dröge
- G. Tezel
- Matthias Schäfer
- I. Bhutto
- Johnny Tang
- R. Kowluru
- Peidu Jiang
- Hideaki Nagase
Papers from M. Nita's perspective
Ordered by how strongly the researchers M. Nita trusts endorse each paper. Same corpus, a different order for every researcher.
- Free radicals in the physiological control of cell function. (Physiological Reviews)
- HIF-1-mediated expression of pyruvate dehydrogenase kinase: a metabolic switch required for cellular adaptation to hypoxia. (Cell Metabolism)
- Inhibition of GAPDH activity by poly(ADP-ribose) polymerase activates three major pathways of hyperglycemic damage in endothelial cells. (Journal of Clinical Investigation)
- The pathobiology of diabetic complications: a unifying mechanism. (Diabetes)
- Oxidative damage–induced inflammation initiates age-related macular degeneration (Nature Medicine)
- The biology of the receptor for advanced glycation end products and its ligands. (Biochimica et Biophysica Acta)
- Protein oxidation and aging (Science)
- Structure and function of matrix metalloproteinases and TIMPs. (Cardiovascular Research)
- Mitochondrial Autophagy Is an HIF-1-dependent Adaptive Metabolic Response to Hypoxia* (Journal of Biological Chemistry)
- The Histone Deacetylase SIRT6 Regulates Glucose Homeostasis via Hif1α (Cell)