Matthew P. Frosch

Physiology · 2001–2019

A researcher in the Science corpus: 19 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 160 citations into it
• Molecular Biology — 140 citations into it
• Psychiatry and Mental health — 76 citations into it
• Cellular and Molecular Neuroscience — 48 citations into it
• Cognitive Neuroscience — 37 citations into it
• Pharmacology — 19 citations into it

Whom Matthew P. Frosch trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. H. Braak
2. D. Selkoe
3. Julie A. Schneider

Papers from Matthew P. Frosch's perspective

Ordered by how strongly the researchers Matthew P. Frosch trusts endorse each paper. Same corpus, a different order for every researcher.

1. Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis (Science)
2. Stages of the Pathologic Process in Alzheimer Disease: Age Categories From 1 to 100 Years (Journal of Neuropathology and Experimental Neurology)
3. Pathways towards and away from Alzheimer's disease (Nature)
4. Neuropathologic features of amnestic mild cognitive impairment. (Archives of Neurology)
5. Role of genes and environments for explaining Alzheimer disease. (Archives of General Psychiatry)
6. Neuropathology of Cognitively Normal Elderly (Journal of Neuropathology and Experimental Neurology)
7. Neuropathology of Alzheimer’s Disease
8. Neuropathologically defined subtypes of Alzheimer's disease with distinct clinical characteristics: A retrospective study (Lancet Neurology)
9. The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeutics (Nature reviews. Drug discovery)
10. Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior. (Behavioural Brain Research)

Their papers

• Single-cell RNA-seq supports a developmental hierarchy in human oligodendroglioma (2016) — 1,053 citations
• Decoupling genetics, lineages, and microenvironment in IDH-mutant gliomas by single-cell RNA-seq (2017) — 879 citations
• Ubiquitination of a New Form of α-Synuclein by Parkin from Human Brain: Implications for Parkinson's Disease (2001) — 1,104 citations
• Common variants in MS4A4/MS4A6E, CD2uAP, CD33, and EPHA1 are associated with late-onset Alzheimer’s disease (2011) — 1,887 citations
• An integrative model of cellular states, plasticity and genetics for glioblastoma (2019) — 2,235 citations
• Genetic meta-analysis of diagnosed Alzheimer’s disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing (2019) — 2,422 citations
• High-resolution magnetic resonance imaging reveals nuclei of the human amygdala: manual segmentation to automatic atlas (2017) — 426 citations
• A computational atlas of the hippocampal formation using ex vivo, ultra-high resolution MRI: Application to adaptive segmentation of in vivo MRI (2015) — 1,221 citations
• Simple, scalable proteomic imaging for high-dimensional profiling of intact systems (2015) — 437 citations
• Validating novel tau PET tracer [F-18]-AV-1451 (T807) on postmortem brain tissue (2015) — 615 citations
• Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature (2012) — 1,882 citations
• National Institute on Aging-Alzheimer’s Association guidelines for the neuropathologic assessment of Alzheimer’s disease: a practical approach (2011) — 2,639 citations
• Neuropathological alterations in Alzheimer disease. (2011) — 3,148 citations
• Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS (2010) — 712 citations
• Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions (2010) — 595 citations
• Characterization of amyloid deposition in the APPswe/PS1dE9 mouse model of Alzheimer disease. (2006) — 733 citations
• Early Aβ accumulation and progressive synaptic loss, gliosis, and tangle formation in AD brain (2004) — 687 citations
• Insulin-degrading enzyme regulates the levels of insulin, amyloid β-protein, and the β-amyloid precursor protein intracellular domain in vivo (2003) — 1,348 citations
• Insulin-degrading enzyme regulates the levels of insulin, amyloid beta-protein, and the beta-amyloid precursor protein intracellular domain in vivo. (2003) — 751 citations