Hubert Serve
Molecular Biology · 2000–2017
A researcher in the Science corpus: 6 papers; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 42 citations into it; #13,324 of 17,773 by trust here, #6,817 by citations
Whom Hubert Serve trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Hubert Serve overall
Papers from Hubert Serve's perspective
Ordered by how strongly the researchers Hubert Serve trusts endorse each paper. Same corpus, a different order for every researcher.
- A role for ubiquitin in selective autophagy. (Molecules and Cells)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- Autophagy fights disease through cellular self-digestion (Nature)
- LC3, GABARAP and GATE16 localize to autophagosomal membrane depending on form-II formation (Journal of Cell Science)
- p62/SQSTM1 Binds Directly to Atg8/LC3 to Facilitate Degradation of Ubiquitinated Protein Aggregates by Autophagy* (Journal of Biological Chemistry)
- Mammalian Atg18 (WIPI2) localizes to omegasome-anchored phagophores and positively regulates LC3 lipidation (Autophagy)
- Atg8 controls phagophore expansion during autophagosome formation. (Molecular Biology of the Cell)
- Mature ribosomes are selectively degraded upon starvation by an autophagy pathway requiring the Ubp3p/Bre5p ubiquitin protease (Nature Cell Biology)
- FoxO3 controls autophagy in skeletal muscle in vivo. (Cell Metabolism)
- Nix is a selective autophagy receptor for mitochondrial clearance (EMBO Reports)
Their papers
- MALAT-1, a novel noncoding RNA, and thymosin β4 predict metastasis and survival in early-stage non-small cell lung cancer (2003) — 2,220 citations
- Ubiquitination and selective autophagy (2012) — 735 citations
- Vascular endothelial growth factor and interleukin-6 in paracrine tumor-stromal cell interactions in multiple myeloma. (2000) — 599 citations
- Midostaurin plus Chemotherapy for Acute Myeloid Leukemia with a FLT3 Mutation (2017) — 1,974 citations
- Analysis of FLT3 length mutations in 1003 patients with acute myeloid leukemia: correlation to cytogenetics, FAB subtype, and prognosis in the AMLCG study and usefulness as a marker for the detection of minimal residual disease. (2002) — 1,024 citations
- Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathways. (2000) — 642 citations