N. Villeneuve

Molecular Biology · 2008–2011

A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom N. Villeneuve trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Donna D. Zhang

Papers from N. Villeneuve's perspective

Ordered by how strongly the researchers N. Villeneuve trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway. (Annual Review of Pharmacology and Toxicology)
  2. Distinct Cysteine Residues in Keap1 Are Required for Keap1-Dependent Ubiquitination of Nrf2 and for Stabilization of Nrf2 by Chemopreventive Agents and Oxidative Stress (Molecular and Cellular Biology)
  3. BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase (Molecular and Cellular Biology)
  4. Keap1-dependent Proteasomal Degradation of Transcription Factor Nrf2 Contributes to the Negative Regulation of Antioxidant Response Element-driven Gene Expression* (Journal of Biological Chemistry)
  5. Direct interaction between Nrf2 and p21(Cip1/WAF1) upregulates the Nrf2-mediated antioxidant response. (Molecules and Cells)
  6. Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice. (Cell)
  7. A role for ubiquitin in selective autophagy. (Molecules and Cells)
  8. Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor (Proceedings of the National Academy of Sciences of the United States of America)
  9. The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (Nature Cell Biology)
  10. Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene. (Journal of Clinical Investigation)

Their papers