Erwin P. Bottinger
Molecular Biology · 2002–2020
A researcher in the Science corpus: 25 papers; trusts 9 researchers and is trusted by 4 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 258 citations into it; #8,482 of 17,773 by trust here, #2,765 by citations
- Endocrinology, Diabetes and Metabolism — 32 citations into it
- Computational Theory and Mathematics — 11 citations into it
Whom Erwin P. Bottinger trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Erwin P. Bottinger overall
Papers from Erwin P. Bottinger's perspective
Ordered by how strongly the researchers Erwin P. Bottinger trusts endorse each paper. Same corpus, a different order for every researcher.
- The transcription factor Snail controls epithelial–mesenchymal transitions by repressing E-cadherin expression (Nature Cell Biology)
- Transforming growth factor beta-1 induces snail transcription factor in epithelial cell lines: mechanisms for epithelial mesenchymal transitions. (Journal of Biological Chemistry)
- The snail superfamily of zinc-finger transcription factors (Nature reviews. Molecular cell biology)
- The transcription factor Snail is a repressor of E-cadherin gene expression in epithelial tumour cells (Nature Cell Biology)
- Epithelial–mesenchymal transitions in tumour progression (Nature Reviews. Cancer)
- Diverse cellular and molecular mechanisms contribute to epithelial plasticity and metastasis (Nature reviews. Molecular cell biology)
- Regulation of the Polarity Protein Par6 by TGFß Receptors Controls Epithelial Cell Plasticity (Science)
- The two-handed E box binding zinc finger protein SIP1 downregulates E-cadherin and induces invasion. (Molecules and Cells)
- The Mouse Snail Gene Encodes a Key Regulator of the Epithelial-Mesenchymal Transition (Molecular and Cellular Biology)
- TGF‐β receptor‐activated p38 MAP kinase mediates Smad‐independent TGF‐β responses
Their papers
- Integration of TGF‐β/Smad and Jagged1/Notch signalling in epithelial‐to‐mesenchymal transition (2004) — 690 citations
- TGF-β and epithelial-to-mesenchymal transitions (2005) — 967 citations
- The genetic architecture of type 2 diabetes (2016) — 1,066 citations
- Identification of type 2 diabetes subgroups through topological analysis of patient similarity (2015) — 544 citations
- A comprehensive 1000 Genomes-based genome-wide association meta-analysis of coronary artery disease (2015) — 2,034 citations
- TGF-β signaling in renal disease (2002) — 679 citations
- Association analyses based on false discovery rate implicate new loci for coronary artery disease (2017) — 631 citations
- Characterising the loss-of-function impact of 5’ untranslated region variants in 15,708 individuals (2019) — 162 citations
- AKI in Hospitalized Patients with COVID-19. (2020) — 606 citations
- A structural variation reference for medical and population genetics (2020) — 876 citations
- The Polygenic and Monogenic Basis of Blood Traits and Diseases (2020) — 602 citations
- Genetic analyses of diverse populations improves discovery for complex traits (2019) — 901 citations
- Evaluating drug targets through human loss-of-function genetic variation (2019) — 168 citations
- Associations of autozygosity with a broad range of human phenotypes (2019) — 112 citations
- A catalog of genetic loci associated with kidney function from analyses of a million individuals (2019) — 744 citations
- Genetic analysis of over one million people identifies 535 new loci associated with blood pressure traits (2018) — 1,148 citations
- An Expanded Genome-Wide Association Study of Type 2 Diabetes in Europeans (2017) — 677 citations
- Systematic Evaluation of Pleiotropy Identifies 6 Further Loci Associated With Coronary Artery Disease (2017) — 244 citations
- Refining the accuracy of validated target identification through coding variant fine-mapping in type 2 diabetes (2017) — 401 citations
- Loss-of-Function Mutations in APOC 3 , Triglycerides , and Coronary Disease (2014) — 861 citations