T. Jiang

Molecular Biology · Molecular and Cellular Biology · 2009–2015

A researcher in the Science corpus: 4 papers; trusts 3 researchers and is trusted by 3 researchers here, as inferred from the citation graph.

Subfields

Whom T. Jiang trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Donna D. Zhang
  2. N. Mizushima
  3. Xiao-jun Wang
  4. S. Waguri
  5. M. Komatsu
  6. Beth Levine

Who trusts T. Jiang overall

  1. H. Forman
  2. K. Davies
  3. Donna D. Zhang

Papers from T. Jiang's perspective

Ordered by how strongly the researchers T. Jiang trusts endorse each paper. Same corpus, a different order for every researcher.

  1. The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (Nature Cell Biology)
  2. A role for ubiquitin in selective autophagy. (Molecules and Cells)
  3. Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer. (Molecules and Cells)
  4. Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway. (Annual Review of Pharmacology and Toxicology)
  5. Erratum: Autophagy suppresses tumorigenesis through elimination of p62 (Cell (2009) 137 (1062-1075))
  6. Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy (Proceedings of the National Academy of Sciences of the United States of America)
  7. Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene. (Journal of Clinical Investigation)
  8. Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor (Proceedings of the National Academy of Sciences of the United States of America)
  9. Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice. (Cell)
  10. Protection against electrophile and oxidant stress by induction of the phase 2 response: Fate of cysteines of the Keap1 sensor modified by inducers (Proceedings of the National Academy of Sciences of the United States of America)

Their papers