D. Moller

Molecular Biology · 2000–2013

A researcher in the Science corpus: 8 papers; trusts 7 researchers and is trusted by 5 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 52 citations into it; #7,059 of 17,773 by trust here, #2,408 by citations
• Physiology — 44 citations into it
• Epidemiology — 30 citations into it
• Endocrine and Autonomic Systems — 12 citations into it

Whom D. Moller trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. A. Berg
2. T. Willson
3. I. Shimomura
4. M. Yuan
5. E. Ford
6. Toshimasa Yamauchi

Who trusts D. Moller overall

1. Alok Kumar
2. David J. Loane
3. Brent E. Wisse
4. D. MacEwan
5. Federica Zatterale
6. Herbert Tilg
7. J. Scott
8. K. Kaufman

Papers from D. Moller's perspective

Ordered by how strongly the researchers D. Moller trusts endorse each paper. Same corpus, a different order for every researcher.

1. Proteolytic cleavage product of 30-kDa adipocyte complement-related protein increases fatty acid oxidation in muscle and causes weight loss in mice. (Proceedings of the National Academy of Sciences of the United States of America)
2. The PPARs: from orphan receptors to drug discovery. (Journal of Medicinal Chemistry)
3. The adipocyte-secreted protein Acrp30 enhances hepatic insulin action (Nature Medicine)
4. Decreased IRS-2 and increased SREBP-1c lead to mixed insulin resistance and sensitivity in livers of lipodystrophic and ob/ob mice. (Molecules and Cells)
5. The fat-derived hormone adiponectin reverses insulin resistance associated with both lipoatrophy and obesity (Nature Medicine)
6. Plasma concentrations of a novel, adipose-specific protein, adiponectin, in type 2 diabetic patients. (Arteriosclerosis, Thrombosis and Vascular Biology)
7. A role for AMP-activated protein kinase in contraction- and hypoxia-regulated glucose transport in skeletal muscle. (Molecules and Cells)
8. Reversal of Obesity- and Diet-Induced Insulin Resistance with Salicylates or Targeted Disruption of Ikkβ (Science)
9. Mechanism by which metformin reduces glucose production in type 2 diabetes. (Diabetes)
10. Prevention of fat-induced insulin resistance by salicylate. (Journal of Clinical Investigation)

Their papers

• Role of AMP-activated protein kinase in mechanism of metformin action. (2001) — 5,645 citations
• The mechanisms of action of PPARs. (2002) — 2,641 citations
• New drug targets for type 2 diabetes and the metabolic syndrome (2001) — 1,074 citations
• Metformin increases AMP-activated protein kinase activity in skeletal muscle of subjects with type 2 diabetes. (2002) — 858 citations
• Fibroblast growth factor 21 corrects obesity in mice. (2008) — 1,024 citations
• The effects of LY2405319, an FGF21 analog, in obese human subjects with type 2 diabetes. (2013) — 854 citations
• Metabolic syndrome: a clinical and molecular perspective. (2005) — 669 citations
• Potential Role of TNF-a in the Pathogenesis of Insulin Resistance and Type 2 Diabetes (2000) — 663 citations