S. Apte
Molecular Biology · with Somerville · 1999–2018
A researcher in the Science corpus: 15 papers; trusts 3 researchers and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 41 citations into it; #13,022 of 17,773 by trust here, #4,657 by citations
- Genetics — 29 citations into it
- Immunology — 22 citations into it
- Immunology and Allergy — 17 citations into it; #81 of 206 by trust here, #73 by citations
- Oncology — 17 citations into it
Whom S. Apte trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts S. Apte overall
Papers from S. Apte's perspective
Ordered by how strongly the researchers S. Apte trusts endorse each paper. Same corpus, a different order for every researcher.
- The ADAMTS metalloproteinases.
- Angiogenesis as a therapeutic target (Nature)
- Corneal avascularity is due to soluble VEGF receptor-1 (Nature)
- Fundamental concepts of the angiogenic process. (Current molecular medicine)
- Efficient activation of the lymphangiogenic growth factor VEGF-C requires the C-terminal domain of VEGF-C and the N-terminal domain of CCBE1
- Large-scale association analyses identifies 13 new susceptibility loci for coronary artery disease (Nature Genetics)
- ADAMs: key components in EGFR signalling and development
- Deletion of active ADAMTS5 prevents cartilage degradation in a murine model of osteoarthritis (Nature)
- The myofibroblast matrix: implications for tissue repair and fibrosis (Journal of Pathology)
- Dysregulation of TGF-beta activation contributes to pathogenesis in Marfan syndrome. (Nature Genetics)
Their papers
- LDL receptor-related protein 5 (LRP5) affects bone accrual and eye development. (2001) — 2,274 citations
- Characterization of ADAMTS-9 and ADAMTS-20 as a Distinct ADAMTS Subfamily Related to Caenorhabditis elegans GON-1* (2003) — 336 citations
- Regulation of ADAMTS9 Secretion and Enzymatic Activity by Its Propeptide* (2007) — 66 citations
- Cell-surface Processing of the Metalloprotease Pro-ADAMTS9 Is Influenced by the Chaperone GRP94/gp96* (2009) — 43 citations
- ADAMTS9 is a cell-autonomously acting, anti-angiogenic metalloprotease expressed by microvascular endothelial cells. (2010) — 107 citations
- ADAMTS3 activity is mandatory for embryonic lymphangiogenesis and regulates placental angiogenesis (2015) — 92 citations
- ADAMTS proteins in human disorders (2018) — 240 citations
- Insights on ADAMTS proteases and ADAMTS-like proteins from mammalian genetics. (2015) — 142 citations
- ADAMTS proteins as modulators of microfibril formation and function (2015) — 152 citations
- ADAMTS10 Protein Interacts with Fibrillin-1 and Promotes Its Deposition in Extracellular Matrix of Cultured Fibroblasts* (2011) — 146 citations
- ADAMTS7B, the Full-length Product of the ADAMTS7 Gene, Is a Chondroitin Sulfate Proteoglycan Containing a Mucin Domain* (2004) — 107 citations
- Discovery and Characterization of a Novel, Widely Expressed Metalloprotease, ADAMTS10, and Its Proteolytic Activation* (2004) — 80 citations
- Procollagen II Amino Propeptide Processing by ADAMTS-3 (2001) — 230 citations
- Impaired endochondral ossification and angiogenesis in mice deficient in membrane-type matrix metalloproteinase I. (2000) — 844 citations
- ADAM-TS5, ADAM-TS6, and ADAM-TS7, Novel Members of a New Family of Zinc Metalloproteases (1999)