An oligonucleotide microchip for genome-wide microRNA profiling in human and mouse tissues.
By Chang-Gong Liu, George A. Calin, Brian Meloon, Nir Gamliel, C. Sevignani, M. Ferracin, C. Dumitru, M. Shimizu, S. Zupo, M. Dono and 4 more. Proceedings of the National Academy of Sciences of the United States of America, 2004
972 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/cde7b10619a54868cfbdacf99d051bc631bb86d7
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cancer Research
- Carlo Maria Croce
- George A. Calin
- Scott M. Hammond
- Marilena V. Iorio
- David P. Bartel
- K. Reddy
- Manel Esteller
- V. Narry Kim
In Molecular Biology
- Carlo Maria Croce
- George A. Calin
- Scott M. Hammond
- David P. Bartel
- V. Narry Kim
- Marilena V. Iorio
- Manel Esteller
- K. Reddy
Citations in the corpus
- Altered Expression of miR-21, miR-31, miR-143 and miR-145 Is Related to Clinicopathologic Features of Colorectal Cancer (2008, methodology): “extremely little information is available regarding their physiological and pathological roles. Until now, the most common method for quantifying miRNA was Northern blotting. A number of different approaches to quantify miRNAs have been described, including microarrays [”
- microRNAs as oncogenes and tumor suppressors. (2007, background): “Several laboratories have modified DNA microarray technology to form miRNA microarray technology (Babak et al., 2004; Barad et al., 2004; Liang et al., 2005; Liu et al., 2004; Nelson et al., 2004a,b; Thomson et al., 2004).”
- Comprehensive analysis of microRNA expression patterns in hepatocellular carcinoma and non-tumorous tissues (2006, background): “Using mammalian microarray technology, comprehensive analysis of miRNA expression will help us better understand the relationship between aberrant gene expression and diseases (Liu et al., 2004; Liang et al., 2005).”
- Targeting of the Bmi-1 oncogene/stem cell renewal factor by microRNA-128 inhibits glioma proliferation and self-renewal. (2008, methodology): “Expression profiling was performed using total RNA with the human/mouse miR expression array (V 4.0) and analyzed as described ( 14 ).”
- Regulation of miRNA expression during neural cell specification (2005, background): “A complete description of miRNA expression during stem cell differentiation will require the application of array technology (Krichevsky et al., 2003; Liu et al., 2004).”
- Real-time quantification of microRNAs by stem–loop RT–PCR (2005, methodology): “Less abundant miRNAs routinely escape detection with technologies such as cloning, northern hybridization (31) and microarray analysis (32,33).”
- Widespread deregulation of microRNA expression in human prostate cancer (2008, background): “Volinia et al. (2006) used total mRNA, which would include precursor miRNAs, for labeling and spotted oligonucleotides that detect both precursor and mature miRNAs (Liu et al., 2004).”
- An overview of microRNAs (2015, methodology): “Early studies were performed using microarray, RT-PCR, and bead-based hybridization (Luminex) platforms, while more recent studies have used NGS-based profiling[117, 162-167].”
- Microarray profiling of microRNAs reveals frequent coexpression with neighboring miRNAs and host genes. (2005, background): “Other systems have recently been described for the expression profiling of miRNAs (Babak et al. 2004; Liu et al. 2004; Miska et al. 2004; Nelson et al. 2004; Thomson et al. 2004).”
- MicroRNA gene expression deregulation in human breast cancer. (2005, methodology): “RNA labeling and hybridization on miRNA microarray chips was done as previously described (10).”
- A microRNA DNA methylation signature for human cancer metastasis (2008, methodology): “miRNA microarray profiling was conducted as described (23).”
- MicroRNA signatures associated with cytogenetics and prognosis in acute myeloid leukemia. (2008, background): “39 In this study, there were few patients with favorable cytogenetics, such as inv(16)[4] and t(15;17)[4].”