Analysis of Drosophila photoreceptor axon guidance in eye-specific mosaics.

By T. Newsome, B. Åsling, Barry J. Dickson. Development, 2000

667 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/ba089353f71fccd976ed1315841216e7b278406d

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Citations in the corpus

• Hippo promotes proliferation arrest and apoptosis in the Salvador/Warts pathway (2003, methodology): “To identify genes that regulate tissue growth, we performed a mutagenesis screen by using the eyFLP syste”
• The TEAD/TEF family of transcription factor Scalloped mediates Hippo signaling in organ size control. (2008, methodology): “, 2006), GMR-Gal4 (Freeman, 1996), ey-Gal4 (Newsome et al., 2000), MS1096 (Wang et al.”
• The tumour-suppressor genes NF2/Merlin and Expanded act through Hippo signalling to regulate cell proliferation and apoptosis (2006, methodology): “Mutant clones were induced using the FLP/FRT syste”
• The art and design of genetic screens: Drosophila melanogaster (2002, unknown): “In addition, several different tricks have been used to select against the non-recombinant cells and the twin-spot clones (the reciprocal product of the recombination event, which inherits two copies of the non-mutagenized chromosome arm) 69,70 .”
• Genetic manipulation of genes and cells in the nervous system of the fruit fly (2011, methodology): “The most widely used driver is an eye specific driver, ey-Flp (Newsome et al., 2000), or ey-GAL4; UAS-Flp (Stowers and Schwarz, 1999).”
• Distinct roles for Drosophila Dicer-1 and Dicer-2 in the siRNA/miRNA silencing pathways. (2004, background): “Such genetic mosaics were constitutively generated due to eye-spe- the right arm of the second chromosome, was a homozygous viable complementation group consisting of 39cific mitotic recombination between heterozygous sister chromosomes (Newsome et al., 2000; Stowers and alleles.”
• The Hippo signaling pathway coordinately regulates cell proliferation and apoptosis by inactivating Yorkie, the Drosophila Homolog of YAP. (2005, methodology): “A full-length yki cDNA a driven by the ubiquitous α-tubulin promoter completely d rescued yki null animals to viable and phenotypically S normal adult flies. t We used eyeless-FLP to selectively remove yki func- a tion in over 90% of the eye disc cells (Newsome et al., 2000).”