Molecular complexity and dynamics of cell-matrix adhesions.
By E. Zamir, Benjamin Geiger. Journal of Cell Science, 2001
1,214 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/866ffbce18b75eab1b9f4602cc7122b050bbcc15
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
- Benjamin Geiger
- J. Parsons
- M. Schwartz
- A. Bershadsky
- M. Srichai
- C. von Mering
- Peer Bork
- Michele A. Wozniak
Citations in the corpus
- Focal adhesion regulation of cell behavior. (2004, background): “Currently, efforts are underway to define subsets of focal complexes and adhesions [11], but it is not yet entirely clear which components of a focal complex distinguish it from a focal adhesion.”
- Live-cell photoactivated localization microscopy of nanoscale adhesion dynamics (2008, background): “to super-resolution live-cell imaging of adhesion complexes (ACs”
- Rac and Rho GTPases in cancer cell motility control (2010, background): “All these proteins can bind adaptor proteins through SH2, SH3 or proline rich domains to recruit actin binding proteins (vinculin, paxillin and a-actinin) as well as regulatory molecules PI3K to focal complexes [39,40].”
- Integrins and extracellular matrix in mechanotransduction. (2010, background): “Initial activation through integrin adhesion to matrix recruits up to around 50 diverse signaling molecules to assemble the focal adhesion complex which is capable of responding to environmental stimuli efficiently.(29,30) Mapping of the integrin adhesome binding and signaling interactions identifie”
- Comparative assessment of large-scale data sets of protein–protein interactions (2002, background): “The proteome is much more dynamic than the genome: it changes during development and in response to external stimuli, and the proteins form large interaction networks, in which they regulate and support each othe”
- Adhesion-dependent cell mechanosensitivity. (2003, background): “…events are executed in the adhesion plaques by several types of enzymes, including protein tyrosine kinases [e.g., focal adhesion kinase (FAK), Src, Fyn, etc.] (Geiger et al. 2001, Zamir & Geiger 2001); protein tyrosine phosphatases [e.g., receptor-like tyrosine phosphataseα (RPTP-α)] (Von A nn u.”
- Myofibroblasts and mechano-regulation of connective tissue remodelling (2002, unknown): “pl?Q9Y490">talin , FAK and tyrosine-phosphorylated protein”
- FAK–Src signalling through paxillin, ERK and MLCK regulates adhesion disassembly (2004, background): “When adhesions mature, they grow in size and molecular complexit”