Central nervous system control of food intake

By M. Schwartz, S. Woods, D. Porte, R. Seeley, D. Baskin. Nature, 2000

6,467 citations recorded by the source corpus.

Subfields: Endocrine and Autonomic Systems, Nutrition and Dietetics, Physiology

View Paper: https://www.semanticscholar.org/paper/7b532c5c43e4903b46d9ebff8be7e878ff3aad57

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In Endocrine and Autonomic Systems

In Nutrition and Dietetics

In Physiology

Citations in the corpus

Gut hormones and the regulation of energy homeostasis (2006, background): “Most obese humans already have high circulating levels of leptin and are resistant to its metabolic effect”
Genes and growth performance in crustacean species : a review of relevant genomic studies in crustaceans and other taxa (2013, background): “tides are critical for initiation and regulation of a diverse array of physiological processes including feeding, reproduction and development (Schwartz et al. 2000; Sweedler et al. 2002).”
Kisspeptins and reproduction: physiological roles and regulatory mechanisms. (2012, background): “These neuropeptide products of the POMC gene are wellrecognized anorectic signals produced by a subset of ARC neurons, whose functional balance with NPY neurons in the same nucleus represents a first-order relay for the transmission of the appetite-controlling actions of long-distance signals, such ”
Gut hormone PYY3-36 physiologically inhibits food intake (2002, background): “jpg" NDATA ITEM> ]> Food intake is regulated by the hypothalamus, including the melanocortin and neuropeptide Y (NPY) systems in the arcuate nucleu”
Ghrelin: structure and function. (2005, background): “It is well accepted that appetite is controlled by the brain and that feeding behavior is regulated by complex mechanisms in the central nervous system, in particular the hypothalamus (70, 207, 256).”
The nature of nutrition: a unifying framework (2011, background): “…modulation of taste receptors (Simpson and Raubenheimer 2000; Newland and Yates 2008; Carleton et al. 2010), andmore centrally as signals from systemic and peripheral sources converge onto the neural circuitry that controls feeding behaviour (Schwartz et al. 2000; Cota et al. 2007).”
New insights into symptoms and neurocircuit function of anorexia nervosa (2009, neutral): “It is likely that many of the starvation-driven endocrine and metabolic changes that result from AN are compensatory and attempt to conserve energy or stimulate hunger and feeding 18 .”
BioAmbients: an abstraction for biological compartments (2004, background): “For further details see the main text. Adapted from [ 20 ].”
Neuronal PTP1B regulates body weight, adiposity and leptin action (2006, background): “Npy and Agrp stimulate food intake and decrease energy expenditure, and are coexpressed in a subset of arcuate neurons, distinct from those that express the anorexigenic neuropeptide precursor pro-opiomelanocortin (Pomc”
Meeting the energetic demands of insect diapause: nutrient storage and utilization. (2007, background): “, insulin-like growth factors) and perhaps reproduction (Schwartz et al., 2000; Woods and Seeley, 2000; Wynne et al., 2005).”
Inhibition of food intake in obese subjects by peptide YY3-36. (2003, neutral): “Gut hormone PYY(3-36) physiologically inhibits food intake.”
Neurobiology of anorexia and bulimia nervosa. (2008, background): “Studies in animals show that neuropeptides, such as CRH, leptin, the endogenous opioids (such as beta-endorphin), and neuropeptide-Y (NPY) modulate feeding behaviors and energy metabolism [43,44].”