The role of oxidative stress in the onset and progression of diabetes and its complications: asummary of a Congress Series sponsored byUNESCO‐MCBN, the American Diabetes Association and the German…

By P. Rösen, P. Nawroth, G. King, W. Möller, H. Tritschler, L. Packer. Diabetes/Metabolism Research Reviews, 2001

977 citations recorded by the source corpus.

Subfields: Biochemistry, Clinical Biochemistry, Physiology

View Paper: https://www.semanticscholar.org/paper/6185d58a4fd087a9649f4bc6bb289b457d0ee6ba

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Clinical Biochemistry

1. I. Goldfine
2. B. Maddux
3. Joseph L. Evans
4. G. Grodsky
5. C. van Dijk
6. T. Berl
7. P. Havel

In Physiology

1. I. Goldfine
2. B. Maddux
3. Joseph L. Evans
4. G. Grodsky
5. C. van Dijk
6. T. Berl
7. P. Havel

Citations in the corpus

• Molecular Mechanisms Linking Oxidative Stress and Diabetes Mellitus (2020, background): “The various biomarkers for oxidative stress in patients with diabetes include malondialdehyde (MDA), total cholesterol, and reactive hydroperoxides (ROOH) [30].”
• Oxidative stress in the placenta (2004, background): “Like preeclampsia, diabetes is also characterized as a state of endothelial dysfunction and reactive nitrogen and oxygen species contribute to the progression of diabetes (Honing et al. 1998; Rosen et al. 2001).”
• Are oxidative stress-activated signaling pathways mediators of insulin resistance and beta-cell dysfunction? (2003, background): “Because of their ability to directly oxidize and damage DNA, protein, and lipid, ROS are believed to play a key direct role in the pathogenesis of late diabetic complications (9,11).”
• Mechanisms of obesity-associated insulin resistance: many choices on the menu. (2007, neutral): “2B; Halliwell 1995; Rosen et al. 2001; Evans et al. 2002).”
• Contribution of polyol pathway to diabetes-induced oxidative stress. (2003, background): “Inappropriate activation of these important regulatory molecules would have deleterious effects on cellular functions, and it is thought to contribute to the pathogenesis of various diabetic complications (5).”
• Endothelial Dysfunction and Diabetes: Effects on Angiogenesis, Vascular Remodeling, and Wound Healing (2012, background): “There are studies that show direct powerful antioxidant properties of dipyridamole which protects NO bioavailability [362, 363].”