Joseph L. Evans

Molecular Biology · 2002–2003

A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

Whom Joseph L. Evans trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. D. Jump
  2. Antonio Ceriello
  3. M. Yuan

Who trusts Joseph L. Evans overall

  1. P. Havel
  2. M. Qatanani
  3. M. Cerf
  4. Ralph DeFronzo
  5. A. Fardet
  6. S. Summers

Papers from Joseph L. Evans's perspective

Ordered by how strongly the researchers Joseph L. Evans trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage (Nature)
  2. Reversal of Obesity- and Diet-Induced Insulin Resistance with Salicylates or Targeted Disruption of Ikkβ (Science)
  3. Prevention of fat-induced insulin resistance by salicylate. (Journal of Clinical Investigation)
  4. Molecular motors
  5. Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation. (Proceedings of the National Academy of Sciences of the United States of America)
  6. The effects of rosiglitazone on insulin sensitivity, lipolysis, and hepatic and skeletal muscle triglyceride content in patients with type 2 diabetes. (Diabetes)
  7. Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* (Journal of Biological Chemistry)
  8. Anti-inflammatory cyclopentenone prostaglandins are direct inhibitors of IκB kinase (Nature)
  9. Leptin Induces Mitochondrial Superoxide Production and Monocyte Chemoattractant Protein-1 Expression in Aortic Endothelial Cells by Increasing Fatty Acid Oxidation via Protein Kinase A* (Journal of Biological Chemistry)
  10. Simvastatin and niacin, antioxidant vitamins, or the combination for the prevention of coronary disease. (New England Journal of Medicine)

Their papers