B. Maddux
Molecular Biology · 2002–2003
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Molecular Biology — 23 citations into it
- Cancer Research — 12 citations into it
- Surgery — 8 citations into it
Whom B. Maddux trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- D. Jump (in Cancer Research)
Who trusts B. Maddux
- P. Havel (in Physiology)
Papers from B. Maddux's perspective
Ordered by how strongly the researchers B. Maddux trusts endorse each paper. Same corpus, a different order for every researcher.
- Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage (Nature)
- Molecular motors
- Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation. (Proceedings of the National Academy of Sciences of the United States of America)
- Reversal of Obesity- and Diet-Induced Insulin Resistance with Salicylates or Targeted Disruption of Ikkβ (Science)
- Prevention of fat-induced insulin resistance by salicylate. (Journal of Clinical Investigation)
- The effects of rosiglitazone on insulin sensitivity, lipolysis, and hepatic and skeletal muscle triglyceride content in patients with type 2 diabetes. (Diabetes)
- Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* (Journal of Biological Chemistry)
- Simvastatin and niacin, antioxidant vitamins, or the combination for the prevention of coronary disease. (New England Journal of Medicine)
- Leptin Induces Mitochondrial Superoxide Production and Monocyte Chemoattractant Protein-1 Expression in Aortic Endothelial Cells by Increasing Fatty Acid Oxidation via Protein Kinase A* (Journal of Biological Chemistry)
- Minireview: Secondary β-Cell Failure in Type 2 Diabetes-A Convergence of Glucotoxicity and Lipotoxicity. (Endocrinology)