Contribution of polyol pathway to diabetes-induced oxidative stress.

By S. Chung, E. Ho, K. Lam, Sookja K. Chung. Journal of the American Society of Nephrology, 2003

611 citations recorded by the source corpus.

Subfields: Cell Biology, Clinical Biochemistry, Physiology

View Paper: https://www.semanticscholar.org/paper/d4eb52c776a1d81c383ae1cafc719355ec31f543

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cell Biology

1. C. van Dijk
2. T. Berl

In Clinical Biochemistry

1. C. van Dijk
2. T. Berl

In Physiology

1. C. van Dijk
2. T. Berl

Citations in the corpus

• A Glimpse of Various Pathogenetic Mechanisms of Diabetic Nephropathy (2011, background): “The relevance of the polyol pathway in diabetic lesions is highlighted by studies in which the eye lens of mice overexpressing AR exhibited reduced levels of GSH (16).”
• Pathogenesis of Diabetic Nephropathy (2004, positive): “Also, fructose and its metabolites when glycosylated are more potent glycation end-products fueling AGE-induced oxidative stress [76].”
• Diabetic Nephropathy: Mechanisms of Renal Disease Progression (2008, background): “The relevance of polyol pathway in diabetic lesions per se is unclear; however, reduced levels of GSH in the eye lens in mice overexpressing AR have been reported (12).”