Vancomycin resistance in gram-positive cocci.
By P. Courvalin. Clinical Infectious Diseases, 2006
1,025 citations recorded by the source corpus.
Subfields: Infectious Diseases, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/270ed7001f3282b72912fd7e79e5e158653400b7
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In Infectious Diseases
In Molecular Biology
- Leonor Sánchez-Busó
- Simon R. Harris
- Andrew J. Page
- T. Foster
- Julian Parkhill
- William R. Miller
- P. Bennett
- Gerard D. Wright
Citations in the corpus
- Mechanisms of antibiotic resistance in enterococci (2014, background): “Resistance to these agents, typified by vancomycin, can be described as high-level (MIC >64 mg/ml) or low-level (MIC between 4 and 32 mg/ml), both due to a change in the terminal amino acids of the peptidoglycan precursor from D-Ala-D-Ala to D-alanine-D-lactate (D-AlaD-Lac) or, less frequently, D-al”
- Plasmid encoded antibiotic resistance: acquisition and transfer of antibiotic resistance genes in bacteria (2008, background): “If the basis of resistance is genetically discrete, that is dependent on a single gene or a few linked genes, rather than the interaction of complex systems, then there is no reason, in principle, why these cannot be mobilized on to plasmids and transferred to other bacteria; witness the somewhat co”
- The antibiotic resistome: the nexus of chemical and genetic diversity (2007, background): “Extensive genetic and biochemical study by the groups of Patrice Courvalin and Christopher Walsh have identified the essential roles of each protein in drug resistanc”
- Mechanisms of Antibiotic Resistance (2016, background): “The reader is referred to other comprehensive reviews for additional details on glycopeptide resistance (13, 74, 76).”
- ARIBA: rapid antimicrobial resistance genotyping directly from sequencing reads (2017, background): “The phenotypic resistance is due to the presence of an operon comprising up to seven genes vanB, vanH, vanR, vanS, vanW, vanX, and vanY [24].”
- Antibiotic resistance in Staphylococcus aureus. Current status and future prospects (2017, background): “lipid IImoleculewithD-lactate replacingD-Ala5 (Courvalin 2006; Zeng et al. 2016).”
- Targeting bacterial membrane function: an underexploited mechanism for treating persistent infections (2010, background): “Lipoglycopeptide resistance can also result from expression of the vanA gene clusters; this expression leads to the production of peptidoglycan precursors ending in D -Ala- D -lactate instead of D -Ala- D -Ala, reducing the binding of glycopeptides to peptidoglycan by up to 1,000-fol”
- Antimicrobial Resistance in Bacteria: Mechanisms, Evolution, and Persistence (2019, background): “…later, in 1979, coagulase-negative staphylococci resistant to vancomycin were reported and ten years later resistance in enterococci was described (Courvalin 2006), followed by, in 1997, the report of less-susceptible S. aureus (vancomycin-intermediate S. aureus, VISA) strains in Japan (Levine…”