NAD+/NADH and NADP+/NADPH in Cellular Functions and Cell Death: Regulation and Biological Consequences

By W. Ying. Antioxidants and Redox Signaling, 2008

1,463 citations recorded by the source corpus.

Subfields: Epidemiology, Geriatrics and Gerontology, Physiology

View Paper: https://www.semanticscholar.org/paper/261ca6ef8b64d583c7ef275702653309b6290b40

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. A. Dinkova-Kostova
2. W. Kraus
3. J. Hayes
4. Johan Auwerx

In Geriatrics and Gerontology

1. Johan Auwerx
2. Keir J. Menzies
3. C. Cantó
4. A. Dinkova-Kostova
5. W. Kraus
6. S. Imai
7. Jun Yoshino
8. Joseph A. Baur

In Physiology

1. Johan Auwerx
2. Keir J. Menzies
3. C. Cantó
4. A. Dinkova-Kostova
5. W. Kraus
6. J. Hayes
7. K. Tew
8. S. Imai

Citations in the corpus

• Oxidative Stress in Cancer (2020, background): “Importantly, NADPH provides reducing equivalents that allow GSH to be generated from GSSG and thioredoxin-(SH)2 to be generated from thioredoxin-S2 by the catalytic actions of glutathione reductase (GSR) and thioredoxin reductases (TXNRD1 and TXNRD2), respectively (Ying, 2008).”
• NAD+ metabolism and the control of energy homeostasis - a balancing act between mitochondria and the nucleus (2015, background): “NAD+ and NADP+ perform similar redox functions within the cell, but the latter is more confined to biosynthetic pathways and redox protective roles (reviewed in Ying, 2008).”
• PARPs and ADP-ribosylation: recent advances linking molecular functions to biological outcomes (2017, background): “These include NADases (NAD+ glycohydrolases), CD38, and sirtuins (Lin 2007; Ying 2008).”