Is job strain a major source of cardiovascular disease risk?

By K. Belkic, P. Landsbergis, P. Schnall, D. Baker. Scandinavian Journal of Work, Environment and Health, 2004

770 citations recorded by the source corpus.

Empirical studies on job strain and cardiovascular disease (CVD), their internal validity, and the likely direction of biases were examined. The 17 longitudinal studies had the highest validity ratings. In all but two, biases towards the null dominated. Eight, including several of the largest, showed significant positive results; three had positive, nonsignificant findings. Six of nine case-control studies had significant positive findings; recall bias leading to overestimation appears to be fairly minimal. Four of eight cross-sectional studies had significant positive results. Men showed strong, consistent evidence of an association between exposure to job strain and CVD. The data of the women were more sparse and less consistent, but, as for the men, most of the studies probably underestimated existing effects. Other elements of causal inference, particularly biological plausibility, corroborated that job strain is a major CVD risk factor. Additional intervention studies are needed to examine the impact of ameliorating job strain upon CVD-related outcomes.

Subfields: General Health Professions

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Citations in the corpus

Testing Karasek's learning and strain hypotheses on young workers in their first job (2007, background): “The two postulated main effects are generally supported: both an increase in job demands and a decrease in the job's decision latitude are associated with an increase in strains and various indicators of ill-health, such as heart and vascular diseases (for reviews, see Belkic, Landsbergis, Schnall, ”
The relationship of autonomic imbalance, heart rate variability and cardiovascular disease risk factors. (2010, background): “The lowest quartile of HF power was associated with incident MI, incident coronary heart disease (CHD), fatal CHD, and fatal non-CHD deaths in those with diabeteswithhazard ratios ranging from1.”
Recovery as an explanatory mechanism in the relation between acute stress reactions and chronic health impairment. (2006, neutral): “The following articles refer to this text: 2006;32(6):413-528; 2006;32(6):413-528; 2008;34(3):165-234; 2008;34(3):165-234; 2008;34(5):323-406; 2009;35(3):157-240; 2012;38(3):187-290; 2012;38(4):291-390; 2013;39(4):321-426; 2013;39(6):531-634; 2014;40(5):437-538; 2015;41(2):107-218; 2015;41(3):219-32”
Work Stress and Employee Health (2013, methodology): “Despite this conservatism, their conclusions about the risks associated with work stress reinforce those from Belkic et al.’s (2004) broader narrative review. Second, covariates controlled in these studies included physiological variables that, from an Al theory perspective, represent secondary medi”
Beyond motivation: job and work design for development, health, ambidexterity, and more. (2014, background): “High demands combined with low control have also been shown to affect cardiovascular disease in a series of rigorous studies, particularly for men (Belkic et al. 2004); an explanation for this effect is that these jobs promote psychological strain, hypertension, and/or physical risk factors like…”