K. Mills

Geriatrics and Gerontology · Cell Metabolism · 2007–2016

A researcher in the Science corpus: 3 papers; trusts 3 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

Whom K. Mills trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. S. Imai
  2. C. Cantó
  3. A. Berg
  4. R. Houtkooper
  5. Seon-Young Kim
  6. Jun Yoshino
  7. Nir Barzilai
  8. S. Shoelson
  9. L. Guarente
  10. M. Hasmann

Who trusts K. Mills overall

  1. Jun Yoshino
  2. C. Cantó
  3. Joseph A. Baur
  4. S. Imai
  5. R. Houtkooper

Papers from K. Mills's perspective

Ordered by how strongly the researchers K. Mills trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Mitonuclear protein imbalance as a conserved longevity mechanism (Nature)
  2. The NAD Biosynthesis Pathway Mediated by Nicotinamide Phosphoribosyltransferase Regulates Sir2 Activity in Mammalian Cells* (Journal of Biological Chemistry)
  3. Nicotinamide mononucleotide, a key NAD+ intermediate, treats the pathophysiology of diet- and age-induced diabetes in mice (Cell Metabolism)
  4. FK866, a highly specific noncompetitive inhibitor of nicotinamide phosphoribosyltransferase, represents a novel mechanism for induction of tumor cell apoptosis. (Cancer Research)
  5. NAD+ metabolism in health and disease.
  6. Glucose restriction inhibits skeletal myoblast differentiation by activating SIRT1 through AMPK-mediated regulation of Nampt. (Developmental Cell)
  7. Transcriptional silencing and longevity protein Sir2 is an NAD-dependent histone deacetylase (Nature)
  8. Interdependence of AMPK and SIRT1 for metabolic adaptation to fasting and exercise in skeletal muscle (Cell Metabolism)
  9. Increased dosage of mammalian Sir2 in pancreatic beta cells enhances glucose-stimulated insulin secretion in mice. (Cell Metabolism)
  10. A phylogenetically conserved NAD+-dependent protein deacetylase activity in the Sir2 protein family. (Proceedings of the National Academy of Sciences of the United States of America)

Their papers