J. Rathmell
Molecular Biology · 2000–2020
A researcher in the Science corpus: 14 papers; trusts 8 researchers and is trusted by 10 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 157 citations into it; #5,394 of 17,773 by trust here, #2,475 by citations
- Immunology — 120 citations into it; #786 of 2,160 by trust here, #253 by citations
- Cancer Research — 67 citations into it; #1,425 of 1,780 by trust here, #399 by citations
- Surgery — 28 citations into it
- Biochemistry — 18 citations into it
- Genetics — 18 citations into it
Whom J. Rathmell trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts J. Rathmell overall
- Heather L. Wieman
- Nancie J. MacIver
- Craig B. Thompson
- Sophia Y. Lunt
- J. Wofford
- Ryan D. Michalek
- Michael D. Buck
- Ralph J. Deberardinis
- W. Zong
- J. Chipuk
Papers from J. Rathmell's perspective
Ordered by how strongly the researchers J. Rathmell trusts endorse each paper. Same corpus, a different order for every researcher.
- The biology of cancer: metabolic reprogramming fuels cell growth and proliferation. (Cell Metabolism)
- Enhancing CD8 T Cell Memory by Modulating Fatty Acid Metabolism (Nature)
- mTOR regulates memory CD8 T cell differentiation (Nature)
- c-Myc suppression of miR-23 enhances mitochondrial glutaminase and glutamine metabolism (Nature)
- AMP-activated protein kinase induces a p53-dependent metabolic checkpoint. (Molecules and Cells)
- The AKT–mTOR axis regulates de novo differentiation of CD4+Foxp3+ cells (Journal of Experimental Medicine)
- Cutting Edge: Distinct Glycolytic and Lipid Oxidative Metabolic Programs Are Essential for Effector and Regulatory CD4+ T Cell Subsets (Journal of Immunology)
- Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake. (Molecular Biology of the Cell)
- Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction (Proceedings of the National Academy of Sciences of the United States of America)
- Akt-Directed Glucose Metabolism Can Prevent Bax Conformation Change and Promote Growth Factor-Independent Survival (Molecular and Cellular Biology)
Their papers
- The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues. (2000) — 1,515 citations
- Akt-Directed Glucose Metabolism Can Prevent Bax Conformation Change and Promote Growth Factor-Independent Survival (2003) — 599 citations
- Cytokine stimulation promotes glucose uptake via phosphatidylinositol-3 kinase/Akt regulation of Glut1 activity and trafficking. (2007) — 602 citations
- Metabolic Regulation of T Lymphocytes (2013) — 1,189 citations
- The Glucose Transporter Glut1 is Selectively Essential for CD4 T Cell Activation and Effector Function (2014) — 1,070 citations
- Glucose Uptake Is Limiting in T Cell Activation and Requires CD28-Mediated Akt-Dependent and Independent Pathways1 (2008) — 814 citations
- Akt1 and Akt2 are required for αβ thymocyte survival and differentiation (2007) — 124 citations
- Targeting Metabolism to Improve the Tumor Microenvironment for Cancer Immunotherapy. (2020) — 830 citations
- A guide to immunometabolism for immunologists (2016) — 2,761 citations
- Phosphoenolpyruvate Is a Metabolic Checkpoint of Anti-tumor T Cell Responses (2015) — 1,313 citations
- Metabolic programming and PDHK1 control CD4+ T cell subsets and inflammation. (2015) — 664 citations
- Guidelines for the use and interpretation of assays for monitoring autophagy (2012) — 4,856 citations
- Cutting Edge: Distinct Glycolytic and Lipid Oxidative Metabolic Programs Are Essential for Effector and Regulatory CD4+ T Cell Subsets (2011) — 1,964 citations
- The CD28 signaling pathway regulates glucose metabolism. (2002) — 1,419 citations