A. Vezzani
Cellular and Molecular Neuroscience · 2003–2020
A researcher in the Science corpus: 12 papers; trusts 9 researchers and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Cellular and Molecular Neuroscience — 66 citations into it; #2,744 of 2,998 by trust here, #1,217 by citations
- Molecular Biology — 66 citations into it
- Neurology — 60 citations into it
- Psychiatry and Mental health — 50 citations into it; #710 of 1,338 by trust here, #485 by citations
- Immunology — 39 citations into it
- Pediatrics, Perinatology and Child Health — 30 citations into it
- Oncology — 22 citations into it
- Biological Psychiatry — 11 citations into it
Whom A. Vezzani trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Robert Dantzer
- C. A. Dinarello
- D. Stellwagen
- B. Viviani
- E. Beattie
- Minh N. H. Nguyen
- William A. Catterall
- N. Abbott
- Shizuo Akira
- Wolfgang Löscher
Who trusts A. Vezzani overall
Papers from A. Vezzani's perspective
Ordered by how strongly the researchers A. Vezzani trusts endorse each paper. Same corpus, a different order for every researcher.
- Control of synaptic strength by glial TNFalpha. (Science)
- Release of chromatin protein HMGB1 by necrotic cells triggers inflammation (Nature)
- CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity (Nature Neuroscience)
- Brain Inflammation in Epilepsy: Experimental and Clinical Evidence (Epilepsia)
- Interleukin-1β Enhances NMDA Receptor-Mediated Intracellular Calcium Increase through Activation of the Src Family of Kinases (Journal of Neuroscience)
- CSF1 receptor signaling is necessary for microglia viability, which unmasks a cell that rapidly repopulates the microglia-depleted adult brain (Neuron)
- Signaling pathways downstream of pattern-recognition receptors and their cross talk. (Annual Review of Biochemistry)
- Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion (EMBO Journal)
- High-Mobility Group Box-1 in Ischemia-Reperfusion Injury of the Heart (Circulation)
- Cytokine, Sickness Behavior, and Depression (Immunology and allergy clinics of North America)
Their papers
- Sox2 deficiency causes neurodegeneration and impaired neurogenesis in the adult mouse brain (2004) — 709 citations
- Drug Resistance in Epilepsy: Clinical Impact, Potential Mechanisms, and New Innovative Treatment Options (2020) — 780 citations
- Neuroinflammatory pathways as treatment targets and biomarkers in epilepsy (2019) — 758 citations
- Epilepsy (2018) — 703 citations
- Neuromodulatory properties of inflammatory cytokines and their impact on neuronal excitability. (2015) — 615 citations
- Glia and epilepsy: excitability and inflammation. (2013) — 832 citations
- The role of inflammation in epilepsy (2010) — 1,820 citations
- The clinico-pathological spectrum of Focal Cortical Dysplasias: a consensus classification proposed by an ad hoc Task Force of the ILAE Diagnostic Methods Commission (2010) — 1,619 citations
- Toll-like receptor 4 and high-mobility group box-1 are involved in ictogenesis and can be targeted to reduce seizures (2010) — 897 citations
- Innate and adaptive immunity during epileptogenesis and spontaneous seizures: evidence from experimental models and human temporal lobe epilepsy. (2008) — 686 citations
- Brain Inflammation in Epilepsy: Experimental and Clinical Evidence (2005) — 1,059 citations
- Interleukin-1β Enhances NMDA Receptor-Mediated Intracellular Calcium Increase through Activation of the Src Family of Kinases (2003) — 846 citations