Glia and epilepsy: excitability and inflammation.

By Orrin Devinsky, A. Vezzani, S. Najjar, N. D. de Lanerolle, M. Rogawski. Trends in Neurosciences, 2013

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International consensus classification of hippocampal sclerosis in temporal lobe epilepsy: A Task Force report from the ILAE Commission on Diagnostic Methods (2013, background): “As evidence grows for glia-mediated excitation and inflammation in modulating or triggering seizures (Devinsky et al., 2013), isolated gliosis may play a pivotal role in the pathophysiology of hippocampal damage, particularly in TLE patients with no-HS.”
The Search for True Numbers of Neurons and Glial Cells in the Human Brain: A Review of 150 Years of Cell Counting (2016, background): “Nevertheless, as shown in Table 6, there now is gradual acceptance of the IF and its conclusions by many neuroscientists and also glia biologists (Brautigam et al., 2012; Devinsky et al., 2013; Streit, 2013; Verkhratsky and Butt, 2013).”
Reactive gliosis and the multicellular response to CNS damage and disease (2014, neutral): “In addition, reactive gliosis of both microglia and astrocytes is a prominent feature of chronically epileptic neural tissue, which can also exhibit BBB breakdown and multicellular inflammation (Devinsky et al., 2013; Jabs et al., 2008; Wetherington et al., 2008).”