Yoshinobu Ichimura
Epidemiology · 2000–2015
A researcher in the Science corpus: 8 papers; trusts 4 researchers and is trusted by 8 researchers here, as inferred from the citation graph.
Subfields
- Epidemiology — 74 citations into it; #389 of 2,148 by trust here, #142 by citations
- Physiology — 11 citations into it
- Public Health, Environmental and Occupational Health — 5 citations into it
Whom Yoshinobu Ichimura trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- A. Vagin
- Zbyszek Otwinowski
- M. Komatsu
- N. Mizushima
- R. Youle
- G. Murshudov
- Beth Levine
- Derek P. Narendra
- Y. Kabeya
- Wladek Minor
Who trusts Yoshinobu Ichimura overall
- D. Klionsky
- N. Mizushima
- Y. Ohsumi
- Gabriele Zaffagnini
- H. Nakatogawa
- Alec C. Kimmelman
- T. Johansen
- Zhifen Yang
- Tomohiro Yorimitsu
- Wen-wen Li
Papers from Yoshinobu Ichimura's perspective
Ordered by how strongly the researchers Yoshinobu Ichimura trusts endorse each paper. Same corpus, a different order for every researcher.
- A role for ubiquitin in selective autophagy. (Molecules and Cells)
- The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (Nature Cell Biology)
- Autophagy fights disease through cellular self-digestion (Nature)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- p62/SQSTM1 Is a Target Gene for Transcription Factor NRF2 and Creates a Positive Feedback Loop by Inducing Antioxidant Response Element-driven Gene Transcription* (Journal of Biological Chemistry)
- Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer. (Molecules and Cells)
- Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy (Proceedings of the National Academy of Sciences of the United States of America)
- Autophagy, Immunity, and Microbial Adaptations (Cell Host and Microbe)
- A Noncanonical Mechanism of Nrf2 Activation by Autophagy Deficiency: Direct Interaction between Keap1 and p62 (Molecular and Cellular Biology)
- Development by self-digestion: molecular mechanisms and biological functions of autophagy. (Developmental Cell)
Their papers
- The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (2010) — 2,354 citations
- Structural Basis for Sorting Mechanism of p62 in Selective Autophagy* (2008) — 791 citations
- Phosphorylation of p62 activates the Keap1-Nrf2 pathway during selective autophagy. (2013) — 1,109 citations
- p62/SQSTM1 functions as a signaling hub and an autophagy adaptor (2015) — 764 citations
- Atg8, a ubiquitin-like protein required for autophagosome formation, mediates membrane tethering and hemifusion. (2007) — 1,228 citations
- The Atg12-Atg5 Conjugate Has a Novel E3-like Activity for Protein Lipidation in Autophagy* (2007) — 1,119 citations
- A ubiquitin-like system mediates protein lipidation (2000) — 2,062 citations
- The Reversible Modification Regulates the Membrane-Binding State of Apg8/Aut7 Essential for Autophagy and the Cytoplasm to Vacuole Targeting Pathway (2000) — 1,009 citations