M. Lieber
Molecular Biology · 2002–2017
A researcher in the Science corpus: 8 papers; trusts 15 researchers and is trusted by 22 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 62 citations into it; #2,438 of 17,773 by trust here, #4,775 by citations
- Oncology — 23 citations into it; #545 of 1,698 by trust here, #859 by citations
- Cancer Research — 18 citations into it
- Immunology — 15 citations into it; #1,346 of 2,160 by trust here, #1,311 by citations
- Genetics — 11 citations into it
- Public Health, Environmental and Occupational Health — 6 citations into it
- Plant Science — 5 citations into it
Whom M. Lieber trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Stephen P. Jackson
- J. Falck
- E. Friedberg
- P. Hastings
- Brandi L. Mahaney
- P. Sung
- David H. Jung
- J. R. Lupski
- C. Carvalho
Who trusts M. Lieber overall
- M. Nassal
- W. Gu
- Stephen P. Jackson
- Feng Zhang
- Z. Mao
- Dheeraj Malhotra
- Brandi L. Mahaney
- J. R. Chapman
- Haifeng Wang
- P. Hallast
Papers from M. Lieber's perspective
Ordered by how strongly the researchers M. Lieber trusts endorse each paper. Same corpus, a different order for every researcher.
- Interfaces Between the Detection, Signaling, and Repair of DNA Damage (Science)
- The Mechanism of Human Nonhomologous DNA End Joining* (Journal of Biological Chemistry)
- Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2). (Cell)
- A DNA replication mechanism for generating nonrecurrent rearrangements associated with genomic disorders. (Cell)
- Structural determinants of phosphoinositide 3-kinase inhibition by wortmannin, LY294002, quercetin, myricetin, and staurosporine. (Molecules and Cells)
- R-loops at immunoglobulin class switch regions in the chromosomes of stimulated B cells (Nature Immunology)
- A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci. (Molecules and Cells)
- ATM and DNA-PK Function Redundantly to Phosphorylate H2AX after Exposure to Ionizing Radiation (Cancer Research)
- Mechanism and regulation of human non-homologous DNA end-joining (Nature reviews. Molecular cell biology)
- ATM Phosphorylates Histone H2AX in Response to DNA Double-strand Breaks* (Journal of Biological Chemistry)
Their papers
- The Mechanism of Human Nonhomologous DNA End Joining* (2008) — 739 citations
- R-loops at immunoglobulin class switch regions in the chromosomes of stimulated B cells (2003) — 760 citations
- Hairpin opening and overhang processing by an Artemis/DNA-dependent protein kinase complex in nonhomologous end joining and V(D)J recombination. (2002) — 1,218 citations
- The Mechanism of Double-Strand DNA Break Repair by the Nonhomologous DNA End Joining Pathway (2010) — 2,827 citations
- Mechanism and regulation of human non-homologous DNA end-joining (2003) — 1,034 citations
- Non-homologous DNA end joining and alternative pathways to double-strand break repair (2017) — 1,479 citations
- The Mechanism of (2010) — 423 citations
- Faculty Opinions recommendation of Okazaki fragment maturation in yeast. I. Distribution of functions between FEN1 AND DNA2. (2003) — 102 citations