J. R. Chapman
Molecular Biology · 2007–2012
A researcher in the Science corpus: 3 papers; trusts 4 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 39 citations into it
- Oncology — 20 citations into it
- Cancer Research — 11 citations into it
Whom J. R. Chapman trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts J. R. Chapman overall
Papers from J. R. Chapman's perspective
Ordered by how strongly the researchers J. R. Chapman trusts endorse each paper. Same corpus, a different order for every researcher.
- Sensing DNA Damage Through ATRIP Recognition of RPA-ssDNA Complexes (Science)
- DNA end resection, homologous recombination and DNA damage checkpoint activation require CDK1 (Nature)
- Interfaces Between the Detection, Signaling, and Repair of DNA Damage (Science)
- 53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers (Nature Structural &Molecular Biology)
- Structural basis for the methylation state-specific recognition of histone H4-K20 by 53BP1 and Crb2 in DNA repair. (Cell)
- 53BP1 inhibits homologous recombination in Brca1-deficient cells by blocking resection of DNA breaks (Cell)
- An ATM-Dependent Transcriptional Silencing Program is Transmitted Through Chromatin in Cis to DNA Double Strand Breaks (Cell)
- RAP80 Targets BRCA1 to Specific Ubiquitin Structures at DNA Damage Sites (Science)
- The DNA damage response: putting checkpoints in perspective (Nature)
- Human CtIP promotes DNA end resection (Nature)
Their papers
- Orchestration of the DNA-damage response by the RNF8 ubiquitin ligase (2007) — 955 citations
- Playing the end game: DNA double-strand break repair pathway choice. (2012) — 1,608 citations
- DNA helicases Sgs1 and BLM promote DNA double-strand break resection. (2008) — 601 citations