S. Rutz
Immunology · 2004–2011
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Immunology — 90 citations into it; #1,645 of 2,160 by trust here, #1,718 by citations
Whom S. Rutz trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts S. Rutz overall
Papers from S. Rutz's perspective
Ordered by how strongly the researchers S. Rutz trusts endorse each paper. Same corpus, a different order for every researcher.
- TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
- Transforming growth factor-beta induces development of the T(H)17 lineage. (Nature)
- Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
- IL-22 increases the innate immunity of tissues. (Immunity)
- Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation (Journal of Experimental Medicine)
- IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
- Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense (Journal of Experimental Medicine)
- Interleukin-10 and the interleukin-10 receptor. (Annual Review of Immunology)
- Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
Their papers
- Regulation and functions of the IL-10 family of cytokines in inflammation and disease. (2011) — 1,887 citations
- Interleukin‐2 is essential for CD4+CD25+ regulatory T cell function (2004) — 620 citations