R. Sumpter

Epidemiology · 2003–2016

A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom R. Sumpter trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. K. Blight
  2. Eileen M. Foy
  3. Masahiro Yamamoto
  4. Katherine A. Fitzgerald
  5. Mitsutoshi Yoneyama

Who trusts R. Sumpter overall

  1. J. Feld
  2. O. Takeuchi
  3. Shizuo Akira
  4. Rashu B. Seth
  5. Taro Kawai
  6. Y. Loo
  7. J. Hoofnagle
  8. M. Gale
  9. Daniel B. Stetson
  10. Lijun Sun

Papers from R. Sumpter's perspective

Ordered by how strongly the researchers R. Sumpter trusts endorse each paper. Same corpus, a different order for every researcher.

  1. The RNA helicase RIG-I has an essential function in double-stranded RNA-induced innate antiviral responses
  2. RICK/Rip2/CARDIAK mediates signalling for receptors of the innate and adaptive immune systems (Nature)
  3. Regulation of Interferon Regulatory Factor-3 by the Hepatitis C Virus Serine Protease (Science)
  4. Inaugural Article: Viral and immunological determinants of hepatitis C virus clearance, persistence, and disease
  5. Distinct and essential roles of transcription factors IRF-3 and IRF-7 in response to viruses for IFN-alpha/beta gene induction. (Immunity)
  6. Cutting Edge: A Novel Toll/IL-1 Receptor Domain-Containing Adapter That Preferentially Activates the IFN-β Promoter in the Toll-Like Receptor Signaling1 (Journal of Immunology)
  7. IRF-3-dependent, NFκB- and JNK-independent activation of the 561 and IFN-β genes in response to double-stranded RNA (Proceedings of the National Academy of Sciences of the United States of America)
  8. Broad activation of the ubiquitin–proteasome system by Parkin is critical for mitophagy (Human Molecular Genetics)
  9. To be or not to be? How selective autophagy and cell death govern cell fate. (Cell)
  10. Control of antiviral defenses through hepatitis C virus disruption of retinoic acid-inducible gene-I signaling. (Proceedings of the National Academy of Sciences of the United States of America)

Their papers