IRF-3-dependent, NFκB- and JNK-independent activation of the 561 and IFN-β genes in response to double-stranded RNA

By K. Peters, Heather L. Smith, G. Stark, G. Sen. Proceedings of the National Academy of Sciences of the United States of America, 2002

143 citations recorded by the source corpus.

Subfields: Cancer Research, Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/46b67b8d7043f76c695d073893f6b0eb68ed113f

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cancer Research

1. Kui Li
2. R. Randall
3. S. Goodbourn

In Immunology

1. Shizuo Akira
2. Y. Loo
3. M. Gale
4. Mitsutoshi Yoneyama
5. T. Fujita
6. R. Randall
7. Eileen M. Foy
8. S. Goodbourn

In Molecular Biology

1. M. Gale
2. T. Fujita
3. Kui Li
4. R. Randall
5. Mitsutoshi Yoneyama
6. S. Goodbourn
7. Y. Loo
8. Shizuo Akira

Citations in the corpus

• Regulating Intracellular Antiviral Defense and Permissiveness to Hepatitis C Virus RNA Replication through a Cellular RNA Helicase, RIG-I (2005, background): “Similarly, IFN treatment induced robust expression of ISG56, a well-defined ISG and IRF3 target gene ( 19 ), in both Huh7 and Huh7.5 cells.”
• Interferons and viruses: an interplay between induction, signalling, antiviral responses and virus countermeasures. (2008, background): “…and c-jun/ATF-2 may not be essential; indeed, IFN induction has been reported under conditions where NFkB or c-jun/ATF-2 are not activated or their binding sites are not required (Goodbourn et al., 1985; Ellis & Goodbourn, 1994; King & Goodbourn, 1994; Peters et al., 2002; Poole et al., 2002).”