W. Mitch
Molecular Biology · 2000–2008
A researcher in the Science corpus: 11 papers; trusts 1 researcher and is trusted by 4 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 29 citations into it
- Epidemiology — 10 citations into it
- Cell Biology — 9 citations into it
Whom W. Mitch trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts W. Mitch overall
Papers from W. Mitch's perspective
Ordered by how strongly the researchers W. Mitch trusts endorse each paper. Same corpus, a different order for every researcher.
- Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy (Proceedings of the National Academy of Sciences of the United States of America)
- Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways (Nature Cell Biology)
- Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo (Nature Cell Biology)
- U Box Proteins as a New Family of Ubiquitin-Protein Ligases* (Journal of Biological Chemistry)
- Functional discovery via a compendium of expression profiles. (Cell)
- An integrated stress response regulates amino acid metabolism and resistance to oxidative stress. (Molecules and Cells)
- Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy. (Cell)
- An important function of Nrf2 in combating oxidative stress: Detoxification of acetaminophen (Proceedings of the National Academy of Sciences of the United States of America)
- Lipodystrophy in the fld mouse results from mutation of a new gene encoding a nuclear protein, lipin (Nature Genetics)
- Regulation of insulin action and pancreatic β-cell function by mutated alleles of the gene encoding forkhead transcription factor Foxo1 (Nature Genetics)
Their papers
- Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression (2004) — 1,572 citations
- Activation of caspase-3 is an initial step triggering accelerated muscle proteolysis in catabolic conditions. (2004) — 736 citations
- Cachexia: a new definition. (2008) — 2,296 citations
- A proposed nomenclature and diagnostic criteria for protein-energy wasting in acute and chronic kidney disease. (2008) — 1,890 citations
- Protein degradation by the ubiquitin-proteasome pathway in normal and disease states. (2006) — 1,185 citations
- Proteinuria, a target for renoprotection in patients with type 2 diabetic nephropathy: lessons from RENAAL. (2004) — 1,004 citations
- Albuminuria, a Therapeutic Target for Cardiovascular Protection in Type 2 Diabetic Patients With Nephropathy (2004) — 770 citations
- Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension. (2003) — 1,686 citations
- Effects of losartan on renal and cardiovascular outcomes in patients with type 2 diabetes and nephropathy. (2001) — 6,186 citations
- AHA Dietary Guidelines: revision 2000: A statement for healthcare professionals from the Nutrition Committee of the American Heart Association. (2000) — 1,532 citations
- AHA Dietary Guidelines: revision 2000: A statement for healthcare professionals from the Nutrition Committee of the American Heart Association. (2000) — 698 citations