P. Jeggo

Molecular Biology · 2003–2015

A researcher in the Science corpus: 9 papers; trusts 7 researchers and is trusted by 5 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 94 citations into it; #7,771 of 17,773 by trust here, #4,105 by citations
• Oncology — 57 citations into it; #1,632 of 1,698 by trust here, #620 by citations
• Cancer Research — 33 citations into it; #724 of 1,780 by trust here, #860 by citations
• Cell Biology — 17 citations into it

Whom P. Jeggo trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. S. West
2. J. Bartek
3. Stephen P. Jackson

Who trusts P. Jeggo overall

1. F. Grin
2. Adrian C. Begg

Papers from P. Jeggo's perspective

Ordered by how strongly the researchers P. Jeggo trusts endorse each paper. Same corpus, a different order for every researcher.

1. ATR disruption leads to chromosomal fragmentation and early embryonic lethality. (Genes & Development)
2. Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks (Nature Cell Biology)
3. ATR and ATRIP: Partners in Checkpoint Signaling (Science)
4. DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis (Nature)
5. MDC1 is a mediator of the mammalian DNA damage checkpoint (Nature)
6. DNA damage-induced G2–M checkpoint activation by histone H2AX and 53BP1 (Nature Cell Biology)
7. Chromatin relaxation in response to DNA double-strand breaks is modulated by a novel ATM- and KAP-1 dependent pathway (Nature Cell Biology)
8. A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage. (Current Biology)
9. Insights into DNA recombination from the structure of a RAD51–BRCA2 complex (Nature)
10. Methylated lysine 79 of histone H3 targets 53BP1 to DNA double-strand breaks (Nature)

Their papers

• ATM and DNA-PK Function Redundantly to Phosphorylate H2AX after Exposure to Ionizing Radiation (2004) — 1,057 citations
• A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci. (2004) — 921 citations
• A splicing mutation affecting expression of ataxia–telangiectasia and Rad3–related protein (ATR) results in Seckel syndrome (2003) — 855 citations
• DNA repair, genome stability and cancer: a historical perspective (2015) — 682 citations
• ATM signaling facilitates repair of DNA double-strand breaks associated with heterochromatin. (2008) — 902 citations
• The role of double-strand break repair — insights from human genetics (2006) — 611 citations
• Mutations in ORC1, encoding the largest subunit of the origin recognition complex, cause microcephalic primordial dwarfism resembling Meier-Gorlin syndrome (2011) — 224 citations
• The impact of a negligent G2/M checkpoint on genomic instability and cancer induction (2007) — 640 citations
• γH2AX foci analysis for monitoring DNA double-strand break repair: Strengths, limitations and optimization (2010) — 637 citations