Tianyan Gao
Molecular Biology · 2005–2007
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 7 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 23 citations into it; #6,350 of 17,773 by trust here, #8,570 by citations
Whom Tianyan Gao trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Tianyan Gao overall
- T. Franke
- Yigong Shi
- B. Manning
- Arkaitz Carracedo
- Ping Wee
- D. Guertin
- A. Newton
- Lewis C. Cantley
- L. Salmena
- R. Shaw
Papers from Tianyan Gao's perspective
Ordered by how strongly the researchers Tianyan Gao trusts endorse each paper. Same corpus, a different order for every researcher.
- Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR Complex (Science)
- Insulin Resistance and a Diabetes Mellitus-Like Syndrome in Mice Lacking the Protein Kinase Akt2 (PKBβ)
- Akt/protein kinase B is constitutively active in non-small cell lung cancer cells and promotes cellular survival and resistance to chemotherapy and radiation. (Cancer Research)
- Insulin resistance and a diabetes mellitus-like syndrome in mice lacking the protein kinase Akt2 (PKB beta). (Science)
- Cytoplasmic localization of p21Cip1/WAF1 by Akt-induced phosphorylation in HER-2/neu-overexpressing cells (Nature Cell Biology)
- The TSC1-2 tumor suppressor controls insulin–PI3K signaling via regulation of IRS proteins (Journal of Cell Biology)
- mTOR inhibition induces upstream receptor tyrosine kinase signaling and activates Akt. (Cancer Research)
- PHLPP: a phosphatase that directly dephosphorylates Akt, promotes apoptosis, and suppresses tumor growth. (Molecules and Cells)
- The phosphatidylinositol 3-Kinase–AKT pathway in human cancer (Nature Reviews. Cancer)
- PKB/Akt: a key mediator of cell proliferation, survival and insulin responses? (Journal of Cell Science)