Yong‐Jing Gao
Physiology · 2008–2014
A researcher in the Science corpus: 3 papers; trusts 5 researchers and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Physiology — 58 citations into it; #1,812 of 2,276 by trust here, #1,405 by citations
- Cellular and Molecular Neuroscience — 28 citations into it; #2,798 of 2,998 by trust here, #2,534 by citations
- Neurology — 26 citations into it; #1,331 of 1,455 by trust here, #845 by citations
- Pharmacology — 22 citations into it
Whom Yong‐Jing Gao trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Yong‐Jing Gao overall
Papers from Yong‐Jing Gao's perspective
Ordered by how strongly the researchers Yong‐Jing Gao trusts endorse each paper. Same corpus, a different order for every researcher.
- p38 Mitogen-Activated Protein Kinase Is Activated after a Spinal Nerve Ligation in Spinal Cord Microglia and Dorsal Root Ganglion Neurons and Contributes to the Generation of Neuropathic Pain (Journal of Neuroscience)
- Glial activation: a driving force for pathological pain. (Trends in Neurosciences)
- The role of neuroinflammation and neuroimmune activation in persistent pain (Pain)
- Minocycline, a Tetracycline Derivative, Is Neuroprotective against Excitotoxicity by Inhibiting Activation and Proliferation of Microglia (Journal of Neuroscience)
- Neuronal plasticity: increasing the gain in pain. (Science)
- Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain (Nature Medicine)
- P2X4 receptors induced in spinal microglia gate tactile allodynia after nerve injury (Nature)
- Microglia as a source and target of cytokines (Glia)
- p38 MAP kinases: key signalling molecules as therapeutic targets for inflammatory diseases (Nature reviews. Drug discovery)
- ERK is sequentially activated in neurons, microglia, and astrocytes by spinal nerve ligation and contributes to mechanical allodynia in this neuropathic pain model (Pain)
Their papers
- Emerging targets in neuroinflammation-driven chronic pain (2014) — 1,004 citations
- Chemokines, neuronal-glial interactions, and central processing of neuropathic pain (2010) — 601 citations
- Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain (2008) — 731 citations