R. Ji
Physiology · 2001–2018
A researcher in the Science corpus: 17 papers; trusts 28 researchers and is trusted by 27 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 196 citations into it; #49 of 2,276 by trust here, #293 by citations
- Cellular and Molecular Neuroscience — 128 citations into it; #813 of 2,998 by trust here, #779 by citations
- Neurology — 89 citations into it; #212 of 1,455 by trust here, #270 by citations
- Molecular Biology — 68 citations into it; #5,674 of 17,773 by trust here, #9,030 by citations
- Pharmacology — 54 citations into it; #73 of 1,112 by trust here, #123 by citations
- Immunology — 37 citations into it
- Sensory Systems — 26 citations into it; #626 of 760 by trust here, #390 by citations
- Nutrition and Dietetics — 23 citations into it
Whom R. Ji trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Clifford J. Woolf
- Linda R. Watkins
- Shan-xue Jin
- Z. Zhuang
- E. Milligan
- M. Tsuda
- U. Hanisch
- B. Hains
- J. Mogil
- K. Moore
Who trusts R. Ji overall
- Clifford J. Woolf
- Alban Latremoliere
- Z. Zhuang
- Jürgen Sandkühler
- S. Cooke
- Shan-xue Jin
- E. Milligan
- J. Campbell
- P. Gerner
- M. Suter
Papers from R. Ji's perspective
Ordered by how strongly the researchers R. Ji trusts endorse each paper. Same corpus, a different order for every researcher.
- Glial activation: a driving force for pathological pain. (Trends in Neurosciences)
- p38 Mitogen-Activated Protein Kinase Is Activated after a Spinal Nerve Ligation in Spinal Cord Microglia and Dorsal Root Ganglion Neurons and Contributes to the Generation of Neuropathic Pain (Journal of Neuroscience)
- The role of neuroinflammation and neuroimmune activation in persistent pain (Pain)
- Minocycline, a Tetracycline Derivative, Is Neuroprotective against Excitotoxicity by Inhibiting Activation and Proliferation of Microglia (Journal of Neuroscience)
- Microglia as a source and target of cytokines (Glia)
- p38 MAPK activation by NGF in primary sensory neurons after inflammation increases TRPV1 levels and maintains heat hyperalgesia. (Neuron)
- Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain (Nature Medicine)
- Neuronal plasticity: increasing the gain in pain. (Science)
- Spinal Glia and Proinflammatory Cytokines Mediate Mirror-Image Neuropathic Pain in Rats (Journal of Neuroscience)
- P2X4 receptors induced in spinal microglia gate tactile allodynia after nerve injury (Nature)
Their papers
- Central sensitization and LTP: do pain and memory share similar mechanisms? (2003) — 1,476 citations
- Neuronal plasticity and signal transduction in nociceptive neurons: implications for the initiation and maintenance of pathological pain. (2001) — 748 citations
- MAP kinase and pain (2008) — 999 citations
- Neuroinflammation and central sensitization in chronic and widespread pain (2018) — 1,320 citations
- Microglia in Pain: Detrimental and protective roles in pathogenesis and resolution of pain (2018) — 668 citations
- Pain Regulation by Non-neuronal Cells and Inflammation (2016) — 1,155 citations
- Different immune cells mediate mechanical pain hypersensitivity in male and female mice (2015) — 1,244 citations
- Emerging targets in neuroinflammation-driven chronic pain (2014) — 1,004 citations
- Glia and pain: Is chronic pain a gliopathy? (2013) — 1,131 citations
- Resolvins RvE1 and RvD1Attenuate Inflammatory Pain via Central and Peripheral Actions (2010) — 601 citations
- Chemokines, neuronal-glial interactions, and central processing of neuropathic pain (2010) — 601 citations
- Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain (2008) — 731 citations
- Cytokine Mechanisms of Central Sensitization: Distinct and Overlapping Role of Interleukin-1β, Interleukin-6, and Tumor Necrosis Factor-α in Regulating Synaptic and Neuronal Activity in the Superficial Spinal Cord (2008) — 1,144 citations
- p38 MAPK, microglial signaling, and neuropathic pain (2007) — 609 citations
- ERK is sequentially activated in neurons, microglia, and astrocytes by spinal nerve ligation and contributes to mechanical allodynia in this neuropathic pain model (2005) — 752 citations
- p38 Mitogen-Activated Protein Kinase Is Activated after a Spinal Nerve Ligation in Spinal Cord Microglia and Dorsal Root Ganglion Neurons and Contributes to the Generation of Neuropathic Pain (2003) — 839 citations
- p38 MAPK activation by NGF in primary sensory neurons after inflammation increases TRPV1 levels and maintains heat hyperalgesia. (2002) — 1,233 citations