Neuronal plasticity: increasing the gain in pain.

By Clifford J. Woolf, M. W. Salter. Science, 2000

3,699 citations recorded by the source corpus.

Subfields: Molecular Biology, Pharmacology, Physiology

View Paper: https://www.semanticscholar.org/paper/7e94f6f9f6d7eebc1f4f16c39ebe5f15250a0d86

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

1. Clifford J. Woolf
2. D. Julius
3. R. Ji
4. R. Kuner
5. Arne May
6. A. Basbaum
7. M. Suter
8. Linda R. Watkins

In Pharmacology

1. Clifford J. Woolf
2. R. Ji
3. Marwan N. Baliki
4. Arne May
5. Linda R. Watkins
6. E. Milligan
7. M. Suter
8. J. Scholz

In Physiology

1. Clifford J. Woolf
2. R. Ji
3. Arne May
4. Linda R. Watkins
5. M. Suter
6. D. Julius
7. Marwan N. Baliki
8. E. Milligan

Citations in the corpus

• The neuro-immune balance in neuropathic pain: involvement of inflammatory immune cells, immune-like glial cells and cytokines. (2010, neutral): “It is not the aim of this article to review the mechanisms of neuropathic pain, and we encourage the reader to see the many excellent reviews on the subject (Costigan et al., 2009b; Latremoliere and Woolf, 2009; Woolf, 2004; Woolf and Salter, 2000).”
• Pathological and protective roles of glia in chronic pain (2009, background): “These well-characterized changes in neuronal and biochemical processing, from normal conditions to a pain-facilitatory state in the dorsal horn of the spinal cord, are collectively known as 'central sensitization' (Ref”
• P2X4 receptors induced in spinal microglia gate tactile allodynia after nerve injury (2003, background): “jpg" NDATA ITEM> ]> Pain after nerve damage is an expression of pathological operation of the nervous syste”
• Can we conquer pain? (2002, background): “Exciting progress is being made in dissecting out the molecular and cellular mechanisms that operate in sensory pathways to generate those neural signals that we ultimately interpret as pai”
• Plasticity of motor systems after incomplete spinal cord injury (2001, background): “They might contribute to the functional reorganization of the spinal circuitry, but they could also be involved in maladaptive changes such as neurogenic pain or spasticit”
• Molecular mechanisms of nociception (2001, background): “Moreover, because expression of neurotransmitters, their receptors and other signalling molecules are dramatically altered after tissue or nerve injur”