A. Gudkov
Molecular Biology · 2003–2008
A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 13 citations into it
- Oncology — 9 citations into it
Whom A. Gudkov trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts A. Gudkov overall
Papers from A. Gudkov's perspective
Ordered by how strongly the researchers A. Gudkov trusts endorse each paper. Same corpus, a different order for every researcher.
- p53AIP1, a potential mediator of p53-dependent apoptosis, and its regulation by Ser-46-phosphorylated p53. (Cell)
- A senescence program controlled by p53 and p16INK4a contributes to the outcome of cancer therapy. (Cell)
- If not apoptosis, then what? Treatment-induced senescence and mitotic catastrophe in tumor cells. (Drug resistance updates)
- Endothelial Apoptosis as the Primary Lesion Initiating Intestinal Radiation Damage in Mice (Science)
- ASPP proteins specifically stimulate the apoptotic function of p53. (Molecules and Cells)
- Apoptosis A Link between Cancer Genetics and Chemotherapy (Cell)
- Cellular senescence as a tumor-suppressor mechanism. (Trends in Cell Biology)
- The INK4a/ARF network in tumour suppression (Nature reviews. Molecular cell biology)
- Senescence is a developmental mechanism that contributes to embryonic growth and patterning. (Cell)
- Endonuclease G is an apoptotic DNase when released from mitochondria (Nature)
Their papers
- The role of p53 in determining sensitivity to radiotherapy (2003) — 607 citations
- An Agonist of Toll-Like Receptor 5 Has Radioprotective Activity in Mouse and Primate Models (2008) — 711 citations