I. Fuss

Immunology · 2002–2015

A researcher in the Science corpus: 10 papers; trusts 3 researchers and is trusted by 6 researchers here, as inferred from the citation graph.

Subfields

Whom I. Fuss trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. W. Strober

Who trusts I. Fuss overall

  1. Q. Guan
  2. W. Strober
  3. Markus F. Neurath
  4. R. Sartor
  5. Y. Iwakura
  6. Christopher A. Hunter
  7. Hasan Zaki

Papers from I. Fuss's perspective

Ordered by how strongly the researchers I. Fuss trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides (Journal of Experimental Medicine)
  2. CD4+CD25+ Regulatory T Cells Can Mediate Suppressor Function in the Absence of Transforming Growth Factor β1 Production and Responsiveness (Journal of Experimental Medicine)
  3. CD4(+)CD25(+) immunoregulatory T cells: gene expression analysis reveals a functional role for the glucocorticoid-induced TNF receptor. (Immunity)
  4. Distinct Functional Lineages of Human Vα24 Natural Killer T Cells (Journal of Experimental Medicine)
  5. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
  6. Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12.
  7. Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation (Journal of Experimental Medicine)
  8. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. (Immunity)
  9. Transforming growth factor-beta induces development of the T(H)17 lineage. (Nature)
  10. Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*

Their papers