Miyuki Sato

Epidemiology · 2012–2021

A researcher in the Science corpus: 3 papers; trusts 5 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom Miyuki Sato trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. D. Selkoe
  2. J. Lemasters
  3. Derek P. Narendra

Papers from Miyuki Sato's perspective

Ordered by how strongly the researchers Miyuki Sato trusts endorse each paper. Same corpus, a different order for every researcher.

  1. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
  2. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  3. The mitochondrial deubiquitinase USP30 opposes parkin-mediated mitophagy (Nature)
  4. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  5. p62/SQSTM1 Binds Directly to Atg8/LC3 to Facilitate Degradation of Ubiquitinated Protein Aggregates by Autophagy* (Journal of Biological Chemistry)
  6. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
  7. Broad activation of the ubiquitin–proteasome system by Parkin is critical for mitophagy (Human Molecular Genetics)
  8. Autophagosome formation from membrane compartments enriched in phosphatidylinositol 3-phosphate and dynamically connected to the endoplasmic reticulum (Journal of Cell Biology)
  9. Defective Hepatic Autophagy in Obesity Promotes ER Stress and Causes Insulin Resistance (Cell Metabolism)
  10. Nix is a selective autophagy receptor for mitochondrial clearance (EMBO Reports)

Their papers