E. Brandt
Journal of Clinical & Cellular Immunology · 2001–2011
A researcher in the Science corpus: 2 papers; trusts 0 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Papers from E. Brandt's perspective
Ordered by how strongly the researchers E. Brandt trusts endorse each paper. Same corpus, a different order for every researcher.
- Production of interleukin 22 but not interleukin 17 by a subset of human skin-homing memory T cells (Nature Immunology)
- IL-22 producing “T22” T-cells account for up-regulated IL-22 in atopic dermatitis (AD), despite reduced IL-17 producing Th17 T-cells (Journal of Allergy and Clinical Immunology)
- Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling. (Journal of Clinical Investigation)
- IL-13 receptors and signaling pathways: an evolving web. (Journal of Allergy and Clinical Immunology)
- IL-25 augments type 2 immune responses by enhancing the expansion and functions of TSLP-DC–activated Th2 memory cells (Journal of Experimental Medicine)
- Interleukin 31, a cytokine produced by activated T cells, induces dermatitis in mice (Nature Immunology)
- Epidermal barrier dysfunction in atopic dermatitis. (Journal of Investigative Dermatology)
- IL-31: a new link between T cells and pruritus in atopic skin inflammation. (Journal of Allergy and Clinical Immunology)
- IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. (Immunity)
- Cytokine modulation of atopic dermatitis filaggrin skin expression. (Journal of Allergy and Clinical Immunology)
Their papers
- Th2 Cytokines and Atopic Dermatitis (2011) — 644 citations
- An etiological role for aeroallergens and eosinophils in experimental esophagitis. (2001) — 620 citations