Robert H. Brown
Neurology · 2006–2017
A researcher in the Science corpus: 9 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Neurology — 106 citations into it
- Genetics — 89 citations into it
- Molecular Biology — 69 citations into it
Whom Robert H. Brown trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- Jeffrey D. Rothstein (in Neurology)
- D. Cleveland (in Neurology)
Papers from Robert H. Brown's perspective
Ordered by how strongly the researchers Robert H. Brown trusts endorse each paper. Same corpus, a different order for every researcher.
- Expanded GGGGCC hexanucleotide repeat in non-coding region of C9ORF72 causes chromosome 9p-linked frontotemporal dementia and amyotrophic lateral sclerosis (Neuron)
- Cell-Free Formation of RNA Granules: Low Complexity Sequence Domains Form Dynamic Fibers Within Hydrogels (Cell)
- Gains or losses: molecular mechanisms of TDP43-mediated neurodegeneration (Nature Reviews Neuroscience)
- Eukaryotic stress granules are cleared by autophagy and Cdc48/VCP function. (Cell)
- Wild-Type Nonneuronal Cells Extend Survival of SOD1 Mutant Motor Neurons in ALS Mice (Science)
- Optineurin is an autophagy receptor for damaged mitochondria in parkin-mediated mitophagy that is disrupted by an ALS-linked mutation (Proceedings of the National Academy of Sciences of the United States of America)
- Mutations in the endosomal ESCRTIII-complex subunit CHMP2B in frontotemporal dementia (Nature Genetics)
- Retrograde Viral Delivery of IGF-1 Prolongs Survival in a Mouse ALS Model (Science)
- Genetic diagnosis by whole exome capture and massively parallel DNA sequencing (Proceedings of the National Academy of Sciences of the United States of America)
- Mutated Human SOD1 Causes Dysfunction of Oxidative Phosphorylation in Mitochondria of Transgenic Mice* (Journal of Biological Chemistry)
Their papers
- ANG mutations segregate with familial and 'sporadic' amyotrophic lateral sclerosis (2006) — 682 citations
- Decoding ALS: from genes to mechanism (2016) — 1,838 citations
- Amyotrophic Lateral Sclerosis. (2017) — 725 citations
- Exome sequencing in amyotrophic lateral sclerosis identifies risk genes and pathways (2015) — 948 citations
- Intrinsic Membrane Hyperexcitability of ALS Patient-Derived Motor Neurons (2014) — 630 citations
- Mutations in the Profilin 1 Gene Cause Familial Amyotrophic Lateral Sclerosis (2012) — 618 citations
- dSarm/Sarm1 Is Required for Activation of an Injury-Induced Axon Death Pathway (2012) — 663 citations
- Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS (2010) — 712 citations
- Molecular biology of amyotrophic lateral sclerosis: insights from genetics (2006) — 1,183 citations