Rafik Amir Salama
Physiology · 2014
A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 52 citations into it
- Molecular Biology — 50 citations into it
- Dermatology — 5 citations into it
Whom Rafik Amir Salama trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- F. d’Adda di Fagagna
- T. Kuilman
- J. Campisi
- C. Michaloglou
- U. Herbig
- Adam Freund
- M. Collado
- Jean-Philippe Coppé
- A. Krtolica
- Y. Sancak
Who trusts Rafik Amir Salama overall
Papers from Rafik Amir Salama's perspective
Ordered by how strongly the researchers Rafik Amir Salama trusts endorse each paper. Same corpus, a different order for every researcher.
- Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
- Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence (Nature Cell Biology)
- Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
- Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Tumour biology: Senescence in premalignant tumours (Nature)
- Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis (Nature)
- Chemokine signaling via the CXCR2 receptor reinforces senescence. (Cell)
Their papers
- Cellular senescence and its effector programs (2014) — 801 citations