Hilmar Bading

Molecular Biology · 2002–2010

A researcher in the Science corpus: 3 papers; trusts 3 researchers and is trusted by 14 researchers here, as inferred from the citation graph.

Subfields

Whom Hilmar Bading trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. G. Hardingham
  2. Dervis A M Salih
  3. A. Tashiro
  4. A. Brunet
  5. J. St-Pierre

Who trusts Hilmar Bading overall

  1. G. Hardingham

Papers from Hilmar Bading's perspective

Ordered by how strongly the researchers Hilmar Bading trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways (Nature Neuroscience)
  2. FoxO transcription factors in the maintenance of cellular homeostasis during aging. (Current Opinion in Cell Biology)
  3. NMDA-receptor-mediated, cell-specific integration of new neurons in adult dentate gyrus
  4. Signaling to the Nucleus by an L-type Calcium Channel-Calmodulin Complex Through the MAP Kinase Pathway (Science)
  5. Suppression of reactive oxygen species and neurodegeneration by the PGC-1 transcriptional coactivators. (Cell)
  6. Thermoregulatory and metabolic defects in Huntington's disease transgenic mice implicate PGC-1alpha in Huntington's disease neurodegeneration. (Cell Metabolism)
  7. NMDA Receptor Subunits Have Differential Roles in Mediating Excitotoxic Neuronal Death Both In Vitro and In Vivo (Journal of Neuroscience)
  8. Insulin-regulated hepatic gluconeogenesis through FOXO1–PGC-1α interaction (Nature)
  9. Increasing p16INK4a expression decreases forebrain progenitors and neurogenesis during ageing (Nature)
  10. Why did NMDA receptor antagonists fail clinical trials for stroke and traumatic brain injury? (Lancet Neurology)

Their papers