E. Chapman
Molecular Biology · with Vega · 2015–2018
A researcher in the Science corpus: 2 papers; trusts 3 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 84 citations into it
- Epidemiology — 30 citations into it
- Immunology — 11 citations into it
Whom E. Chapman trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- E. White (in Epidemiology)
- Donna D. Zhang (in Molecular Biology)
- Toren Finkel (in Molecular Biology)
Papers from E. Chapman's perspective
Ordered by how strongly the researchers E. Chapman trusts endorse each paper. Same corpus, a different order for every researcher.
- Autophagy in the pathogenesis of disease. (Cell)
- Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice. (Cell)
- p62/SQSTM1 Is a Target Gene for Transcription Factor NRF2 and Creates a Positive Feedback Loop by Inducing Antioxidant Response Element-driven Gene Transcription* (Journal of Biological Chemistry)
- Autophagy promotes tumor cell survival and restricts necrosis, inflammation, and tumorigenesis. (Cancer Cell)
- Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice (Journal of Cell Biology)
- Keap1 Recruits Neh2 through Binding to ETGE and DLG Motifs: Characterization of the Two-Site Molecular Recognition Model (Molecular and Cellular Biology)
- Direct interaction between Nrf2 and p21(Cip1/WAF1) upregulates the Nrf2-mediated antioxidant response. (Molecules and Cells)
- Autophagy: process and function. (Genes & Development)
- Loss of Keap1 function activates Nrf2 and provides advantages for lung cancer cell growth. (Cancer Research)
- Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells (Journal of Cell Biology)
Their papers
- NRF2 and the Hallmarks of Cancer. (2018) — 1,479 citations
- p62 links autophagy and Nrf2 signaling. (2015) — 598 citations