Jürgen Sandkühler

Physiology · 2009

A researcher in the Science corpus: 1 paper; trusts 17 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 33 citations into it; #648 of 2,276 by trust here, #1,906 by citations
• Cellular and Molecular Neuroscience — 14 citations into it; #1,228 of 2,998 by trust here, #2,534 by citations
• Neurology — 12 citations into it
• Pharmacology — 12 citations into it
• Cognitive Neuroscience — 8 citations into it
• Molecular Biology — 5 citations into it

Whom Jürgen Sandkühler trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. R. Ji
2. Clifford J. Woolf
3. M. Caterina
4. M. Tsuda
5. N. Bonini
6. E. Milligan
7. Z. Zhuang
8. Irene Tracey
9. T. Bliss
10. Linda R. Watkins

Who trusts Jürgen Sandkühler overall

1. R. Kuner
2. A. Todd
3. George M. Slavich
4. E. Old
5. J. Deuis

Papers from Jürgen Sandkühler's perspective

Ordered by how strongly the researchers Jürgen Sandkühler trusts endorse each paper. Same corpus, a different order for every researcher.

1. Glial activation: a driving force for pathological pain. (Trends in Neurosciences)
2. p38 Mitogen-Activated Protein Kinase Is Activated after a Spinal Nerve Ligation in Spinal Cord Microglia and Dorsal Root Ganglion Neurons and Contributes to the Generation of Neuropathic Pain (Journal of Neuroscience)
3. P2X4 receptors induced in spinal microglia gate tactile allodynia after nerve injury (Nature)
4. Spinal Glia and Proinflammatory Cytokines Mediate Mirror-Image Neuropathic Pain in Rats (Journal of Neuroscience)
5. Partial Peripheral Nerve Injury Promotes a Selective Loss of GABAergic Inhibition in the Superficial Dorsal Horn of the Spinal Cord (Journal of Neuroscience)
6. ERK is sequentially activated in neurons, microglia, and astrocytes by spinal nerve ligation and contributes to mechanical allodynia in this neuropathic pain model (Pain)
7. Role of the Immune system in chronic pain (Nature Reviews Neuroscience)
8. Trans-synaptic shift in anion gradient in spinal lamina I neurons as a mechanism of neuropathic pain (Nature)
9. The CNS role of Toll-like receptor 4 in innate neuroimmunity and painful neuropathy. (Proceedings of the National Academy of Sciences of the United States of America)
10. Cytokine Mechanisms of Central Sensitization: Distinct and Overlapping Role of Interleukin-1β, Interleukin-6, and Tumor Necrosis Factor-α in Regulating Synaptic and Neuronal Activity in the… (Journal of Neuroscience)

Their papers

• Models and mechanisms of hyperalgesia and allodynia. (2009) — 1,042 citations