Bruno Dubois

Physiology · 2003–2021

A researcher in the Science corpus: 25 papers; trusts 6 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom Bruno Dubois trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Clifford R. Jack Jr.
  2. Kaj Blennow
  3. H. Braak
  4. Harald Hampel
  5. Reisa A. Sperling
  6. Wolfram Schultz
  7. Ronald C. Petersen
  8. Randy L. Buckner
  9. E. T. Rolls
  10. Wayne C. Drevets

Who trusts Bruno Dubois overall

  1. Clifford R. Jack Jr.
  2. Kaj Blennow
  3. Michael G. Erkkinen
  4. Henrik Zetterberg
  5. Perminder S. Sachdev
  6. Matthias L. Schroeter
  7. Jia-Hao Sun
  8. Reisa A. Sperling
  9. Emilie Gerardin
  10. Marilyn S. Albert

Papers from Bruno Dubois's perspective

Ordered by how strongly the researchers Bruno Dubois trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Molecular, Structural, and Functional Characterization of Alzheimer's Disease: Evidence for a Relationship between Default Activity, Amyloid, and Memory (Journal of Neuroscience)
  2. Current concepts in mild cognitive impairment. (Archives of Neurology)
  3. Biomarkers for Alzheimer's disease: academic, industry and regulatory perspectives (Nature reviews. Drug discovery)
  4. Hypothetical model of dynamic biomarkers of the Alzheimer’s pathological cascade (Lancet Neurology)
  5. The pathological process underlying Alzheimer’s disease in individuals under thirty (Acta Neuropathologica)
  6. Primary age-related tauopathy (PART): a common pathology associated with human aging (Acta Neuropathologica)
  7. Classification of primary progressive aphasia and its variants (Neurology)
  8. The Consortium to Establish a Registry for Alzheimer's Disease (CERAD)
  9. Clinical and pathological diagnosis of frontotemporal dementia: report of the Work Group on Frontotemporal Dementia and Pick's Disease. (Archives of Neurology)
  10. Cerebrospinal fluid {beta}-amyloid 42 and tau proteins as biomarkers of Alzheimer-type pathologic changes in the brain. (Archives of Neurology)

Their papers