Clinical and pathological diagnosis of frontotemporal dementia: report of the Work Group on Frontotemporal Dementia and Pick's Disease.
By G. Mckhann, M. Albert, Murray C. Grossman, B. Miller, Dennis W. Dickson, John Q. Trojanowski. Archives of Neurology, 2001
1,460 citations recorded by the source corpus.
Subfields: Molecular Biology, Neurology, Physiology
View Paper: https://www.semanticscholar.org/paper/027cd7d3548eae83a7b465d56e2234923ae5dac4
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In Molecular Biology
- D. Holtzman
- John C. Morris
- Rudolph E. Tanzi
- John Q. Trojanowski
- William W. Seeley
- C. Duyckaerts
- Rosa Rademakers
- Dennis W. Dickson
In Neurology
- Dennis W. Dickson
- V. Lee
- John Q. Trojanowski
- D. Holtzman
- William W. Seeley
- John C. Morris
- Ronald C. Petersen
- Christos Davatzikos
In Physiology
- D. Holtzman
- Clifford R. Jack Jr.
- John C. Morris
- Ronald C. Petersen
- Christos Davatzikos
- John Q. Trojanowski
- Dennis W. Dickson
- Rudolph E. Tanzi
Citations in the corpus
- TDP‐43 immunoreactivity in hippocampal sclerosis and Alzheimer's disease (2007, background): “Frontotemporal lobar degeneration (FTLD) is a heterogeneous group of neurodegenerative disorders that are subdivided into tau-positive and -negative disorders according to whether there are tau-immunoreactive neuronal and glial lesions and abnormal conformers of tau protein on biochemical analyses.(”
- The genetic epidemiology of neurodegenerative disease. (2005, background): “FTD is a heterogenous group of syndromes defined clinically by a gradual and progressive change in behavior and personal conduct and/or by a gradual and progressive language dysfunction (62).”
- Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia. (2011, background): “Three sets of bvFTD diagnostic criteria have been published over the past two decades that reflect our evolving knowledge about the presentation and progression of the disease (Brun et al., 1994; Neary et al., 1998; McKhann et al., 2001).”
- Neuropathologic diagnostic and nosologic criteria for frontotemporal lobar degeneration: consensus of the Consortium for Frontotemporal Lobar Degeneration (2007, methodology): “[53], and take into account more recent descriptions of novel disease entities [6, 14], the discovery of causative gene defects (PGRN, VCP, CHMP2B) and linkage to chro-”
- The evolution and pathology of frontotemporal dementia. (2005, background): “A work group on FTD and Pick’s disease (McKhann et al., 2001) included CBD and PSP among the pathological substrates of the clinical syndromes of FTD, recognizing that each pathological entity can manifest itself with either language or behaviour disorder, thus incorporating the basic claims of the…”
- Clinicopathological and imaging correlates of progressive aphasia and apraxia of speech. (2006, methodology): “One case had typical features of Pick's disease (McKhann et al., 2001; Dickson, 1998).”
- Automatic classification of MR scans in Alzheimer’s disease (2008, background): “Because the mix of scanners used was different for normal elderly controls and FTLD subjects, the same four AD subjects were included for comparison between AD and FTLD subjects of group IV to maintain an equal balance of scanners between groups.”
- Inverse relation between in vivo amyloid imaging load and cerebrospinal fluid Aβ42 in humans (2006, background): “One subject was diagnosed with frontotemporal lobar degeneration, a group of disorders characterized, in part, by the absence of cerebral amyloid deposition.(16) The other subject had received a CDR score of 0.”