Fei Zhao
Molecular Biology · with Zhang · 2008–2010
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 26 citations into it
Whom Fei Zhao trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Fei Zhao's perspective
Ordered by how strongly the researchers Fei Zhao trusts endorse each paper. Same corpus, a different order for every researcher.
- Distinct Cysteine Residues in Keap1 Are Required for Keap1-Dependent Ubiquitination of Nrf2 and for Stabilization of Nrf2 by Chemopreventive Agents and Oxidative Stress (Molecular and Cellular Biology)
- A role for ubiquitin in selective autophagy. (Molecules and Cells)
- Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor (Proceedings of the National Academy of Sciences of the United States of America)
- Keap1-dependent Proteasomal Degradation of Transcription Factor Nrf2 Contributes to the Negative Regulation of Antioxidant Response Element-driven Gene Expression* (Journal of Biological Chemistry)
- BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase (Molecular and Cellular Biology)
- Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene. (Journal of Clinical Investigation)
- Erratum: Autophagy suppresses tumorigenesis through elimination of p62 (Cell (2009) 137 (1062-1075))
- The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (Nature Cell Biology)
- Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice. (Cell)
- Autophagy: molecular machinery for self-eating (Cell Death and Differentiation)