S. Giordano
Molecular Biology · Cancer Cell · 2003–2016
A researcher in the Science corpus: 4 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 46 citations into it
Whom S. Giordano trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- Masayuki Yamamoto (in Molecular Biology)
- Donna D. Zhang (in Molecular Biology)
Papers from S. Giordano's perspective
Ordered by how strongly the researchers S. Giordano trusts endorse each paper. Same corpus, a different order for every researcher.
- Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
- Loss of Keap1 function activates Nrf2 and provides advantages for lung cancer cell growth. (Cancer Research)
- Structural basis for defects of Keap1 activity provoked by its point mutations in lung cancer. (Molecules and Cells)
- Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis (Nature)
- Nrf2-Keap1 defines a physiologically important stress response mechanism. (Trends in Molecular Medicine)
- Met, metastasis, motility and more (Nature reviews. Molecular cell biology)
- Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy (Proceedings of the National Academy of Sciences of the United States of America)
- HIF-1, O(2), and the 3 PHDs: how animal cells signal hypoxia to the nucleus. (Cell)
- Scatter-factor and semaphorin receptors: cell signalling for invasive growth (Nature Reviews. Cancer)
- Hypoxia — a key regulatory factor in tumour growth (Nature Reviews. Cancer)
Their papers
- Drug development of MET inhibitors: targeting oncogene addiction and expedience (2008) — 845 citations
- The Dual Roles of NRF2 in Cancer. (2016) — 742 citations
- Amplification of the MET receptor drives resistance to anti-EGFR therapies in colorectal cancer (2013) — 633 citations
- Hypoxia promotes invasive growth by transcriptional activation of the met protooncogene. (2003) — 1,357 citations