J. Götz

Physiology · 2001–2011

A researcher in the Science corpus: 4 papers; trusts 9 researchers and is trusted by 7 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 70 citations into it; #559 of 2,276 by trust here, #537 by citations
• Molecular Biology — 42 citations into it; #6,919 of 17,773 by trust here, #6,535 by citations
• Cellular and Molecular Neuroscience — 33 citations into it; #1,589 of 2,998 by trust here, #741 by citations
• Neurology — 27 citations into it
• Pharmacology — 13 citations into it
• Computational Theory and Mathematics — 7 citations into it; #10,999 of 12,182 by trust here, #6,798 by citations

Whom J. Götz trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. D. Selkoe
2. R. Kayed
3. Lars Bertram
4. M. Goedert
5. V. Lee
6. N. Hirokawa

Who trusts J. Götz overall

1. L. Ittner
2. M. Murphy
3. J. Long
4. M. Kirkitadze
5. D. Holtzman
6. Se Hoon Choi
7. H. Levine
8. E. Mandelkow
9. D. Selkoe
10. Eleanor Drummond

Papers from J. Götz's perspective

Ordered by how strongly the researchers J. Götz trusts endorse each paper. Same corpus, a different order for every researcher.

1. Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function (Science)
2. Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis (Science)
3. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
4. Tauopathy in Drosophila: Neurodegeneration Without Neurofibrillary Tangles (Science)
5. Alzheimer's Disease Is a Synaptic Failure (Science)
6. Tau-mediated neurodegeneration in Alzheimer's disease and related disorders (Nature Reviews Neuroscience)
7. Neuropathologic diagnostic and nosologic criteria for frontotemporal lobar degeneration: consensus of the Consortium for Frontotemporal Lobar Degeneration (Acta Neuropathologica)
8. Inhibition of glycogen synthase kinase-3 by lithium correlates with reduced tauopathy and degeneration in vivo. (Proceedings of the National Academy of Sciences of the United States of America)
9. Clusters of Hyperactive Neurons Near Amyloid Plaques in a Mouse Model of Alzheimer's Disease (Science)
10. Axonopathy and Transport Deficits Early in the Pathogenesis of Alzheimer's Disease (Science)

Their papers

• Formation of Neurofibrillary Tangles in P301L Tau Transgenic Mice Induced by Aβ42 Fibrils (2001) — 1,583 citations
• Animal models of Alzheimer's disease and frontotemporal dementia (2008) — 709 citations
• Amyloid-β and tau — a toxic pas de deux in Alzheimer's disease (2011) — 1,298 citations
• Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's disease mouse models. (2010) — 1,853 citations