Amyloid-β and tau — a toxic pas de deux in Alzheimer's disease
By L. Ittner, J. Götz. Nature Reviews Neuroscience, 2011
1,298 citations recorded by the source corpus.
Subfields: Molecular Biology, Pharmacology, Physiology
View Paper: https://www.semanticscholar.org/paper/45e57fff147fed9a120ab23c752c0111a3eb9dcc
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Citations in the corpus
- Soluble amyloid β-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration (2011, background): “However, there are numerous different ways in which an interaction between Aβ oligomers and tau could occur in AD brains (42), and clarifying precisely how this occurs is the next major step for the approach we report here.”